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Cardiovascular Disorder

This document provides an outline of key topics related to cardiovascular disorders, including heart rate, rhythm, sounds, murmurs, blood pressure, pulse, risk factors for atherosclerotic heart disease, hypertension classifications and complications. Specific assessment techniques are described for inspection, palpation, auscultation and blood pressure measurement. Common antihypertensive medications are also listed.

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Clara De Guzman
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0% found this document useful (0 votes)
374 views

Cardiovascular Disorder

This document provides an outline of key topics related to cardiovascular disorders, including heart rate, rhythm, sounds, murmurs, blood pressure, pulse, risk factors for atherosclerotic heart disease, hypertension classifications and complications. Specific assessment techniques are described for inspection, palpation, auscultation and blood pressure measurement. Common antihypertensive medications are also listed.

Uploaded by

Clara De Guzman
Copyright
© Attribution Non-Commercial (BY-NC)
We take content rights seriously. If you suspect this is your content, claim it here.
Available Formats
Download as DOC, PDF, TXT or read online on Scribd
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CARDIOVASCULAR DISORDER OUTLINE • Heart rate

• Rhythm
Heart Facts • Heart Sounds S1 (lub) and S2 (dub)
• One in six men and one in seven women aged 45-64 has • S1 is first heart sound from closure of Mitral and Tricuspid
some form of heart disease or stroke; the ratio climbs to • S2 is second sound from closure of aortic and pulmonic
one in three at age 65 and over. • Don’t worry about correctly labeling sounds as S3 or S4
• Twice as many people, both men and women, die of for now
cardiovascular disease as die of cancer. • Murmurs are swishing sounds in-between heart sounds
• An estimated 3,150,000 Americans have angina pectoris (Lub-swish-Dub)
• Rheumatic heart disease afflicts 1,340,000 Americans. • Pericardial friction rub - rough, grating sound from
(Data from the American Heart Association's 1994 inflamed pericardial sac
Statistical Supplement, "Heart and Stroke Facts") • Bruit - murmur heard outside of the heart. May be heard
in carotid, jugular, temporal, abdominal, aortic, renal and
Atherosclerosis and Arteriosclerosis femoral arteries
• Common disorder of the arteries. • Take B/P in both arms, lying, sitting and standing
o Fat, cholesterol, and other substances collect in the Pulse Assessment
walls of arteries. • Note whether the pulse is regular or irregular:
o Larger accumulations are called atheromas or o Regular - evenly spaced, may vary slightly with
plaques. respiration
• Fatty tissue erodes wall of the artery, diminishes the o Regularly Irregular - regular pattern overall with
elasticity and interferes with blood flow. "skipped" beats
• Arteriosclerosis (commonly called hardening of the o Irregularly Irregular - chaotic, no real pattern, very
arteries) frequently accompanies atherosclerosis and is difficult to measure rate accurately
not clearly separated from it. o A pulse greater than 100 beats/minute is tachycardia.
o Arteriosclerosis involves deposits along the arteries, Pulse less than 60 beats/minute is bradycardia.
which often contain calcium. o Tachycardia and bradycardia are not necessarily
abnormal. Athletes tend to be bradycardic at rest
Arteriosclerosis and Ateriosclerotic Heart Disease o Tachycardia is a normal response to stress or exercise.
(ASHD) B/P Assessment
• Primarily affects middle age and older • Position and support the arm slightly flexed at the elbow
• Major cause of death in men age 35-44 years old • Center the bladder of the cuff over the brachial artery 2
• Major cause of death in women after menopause cm above the anticubital fold
• Prevention begins at birth • Inflate the cuff until the radial pulse disappears. Inflate
• 3 Main Risk Factors the cuff to 30 mmHg above the estimated systolic
o Smoking, High B/P and High Cholesterol pressure, release slowly.
• Intermittent claudication (pain in calf or leg brought on by • Systolic Pressure - When you start to hear beats
exercise and relieved by rest) is most common early sign • Diastolic pressure when the sounds muffle and disappear.
of arteriosclerosis • Don’t use too small a cuff. The pressure will be 10, 20,
Risk Factors for ASHD even 50 mmHg too high!
• Non-Modifiable • Maximum Cuff Pressure - When the baseline is known or
o Age hypertension is not suspected, it is acceptable in adults to
o Gender inflate to 200 mmHg
o Race • Be aware that there could be an ausculatory gap (a silent
o Family history interval between the true systolic and diastolic
o other illness (diabetic) pressures).
• Minor Factors
o Personality type Blood Pressure Classification in Adults
o Sedentary living Hypertension
o Stress • Consistent elevation of diastolic B/P above 90
o Oral Contraceptive Use • Affects over 58 million people
• Modifiable o Incidence increases with age
o Smoking o Almost twice as prevalent in Blacks
o High calorie, fat, cholesterol, sugar and sodium diet o More severe in Blacks
o High serum lipids • Two Main types - Primary (Essential) hypertension and
o Hypertension Secondary Hypertension
o Obesity o Primary accounts for 90% of all Hypertension
o Lifestyle o Secondary is from a known cause such as
glomerulonephritis, Cushing’s or renal stenosis
Cardiovascular Assessment - Inspection Isolated Systolic Hypertension
• Skin Color (pallor, cyanosis) • Frequently seen in older adults
• Neck vein distention - reflects right atrial pressure • Systolic over 90, with normal diastolic
• Respiratory (dyspnea, Orthopnea) • While commonly ignored, this condition should be
• Location of PMI - if too low indicates enlarged heart treated.
• Presence of edema - fluid volume overload • Studies show that lowering the systolic number cuts down
o Be sure to check sacrum for those on bedrest on strokes and heart attacks in people age 60 and over.
• Nail clubbing - sign of chronic hypoxia Primary Hypertension
• Capillary filling - measure of peripheral circulation • Possible contributing mechanisms:
• Lack of hair on legs/feet o heredity
• Venous stasis or Arterial Ulcers o vascular hypertrophy
• Vericose Veins o defects in ion handling
Palpation o hyperinsulinemia
• PMI (Located at 5th intercostal space, Left MCL) o renin/angiotensin
• Thrills (palpable murmur) thrusts/heaves Malignant Hypertension
• Peripheral pulses - Check all bilateral and compare • Severe, rapidly progressive rise in B/P that damages
• Temperature - check bilateral small arterioles of major organ systems
• Capillary refill - measure of peripheral circulation • Inflammation of the arterioles of the eye is classic finding
Auscultation • Primarily found in black men under 40
• Unless aggressively treated, is rapidly fatal • Renin actives Angiotensinogen which becomes
Angiotensin I and II (with the help of ACE (angiotensin
Complications of Hypertension converting enzyme). Angiotensin II causes
• Atherosclerosis vasoconstriction of blood vessels and stimulates
o Athlerosclerotic vascular occlusion aldosterone
a) coronary artery disease • Aldosterone leads to increased fluid and sodium retention
b) occlusive cerebrovascular • All act to Increase Blood pressure
c) peripheral arterial insufficiency Angiotensinogen Converting Enzyme (ACE) Inhibitors
• aortic dissection • Commonly known as ACE inhibitors
• nephrosclerosis o Captopril (Capoten); Enalapril (Vasotec); Lisinopril
• hypertensive heart disease with cardiac hypertrophy and (Zestril)
congestive heart failure • Used for: Chronic hypertension, congestive heart failure.
• cerebral hemorrhage • Side Effects: Cough, diarrhea, headache, loss of taste,
Antihypertensive Medications nausea, unusual tiredness.
• Diuretics • Report symptoms of
o Thiazide/Thiazide-like diuretics; Loop Diuretics; o dizziness; fainting; skin rash; chest pain.
Potassium Sparing Diuretics Prevention of ASHD
• Adrenergic Inhibitors • DON’T SMOKE
o Beta-Blockers; Centrally Acting and Peripherally • Exercise and weight control
acting Alpha Blockers; Combined Alpha-Beta Blockers o Lowers total cholesterol, LDL's (bad cholesterol) and
• Vasodilators the Triglycerides and may raise the good cholesterol
• ACE Inhibitors (HDL).
• Calcium Antagonists o Eat a low-fat diet and become a semi-vegetarian, eat
Diuretic Treatment red meat/cheese less often
• Potassium Wasting Diuretics o Eat fish (not shell-fish, not fried) once a week
o Thiazide Diuretics - Chlorothiazide (Diuril) and • Increase your intake of soluble fiber
Hydrochlorothiazide (Hyrodiuril) o Oats, beans, barley, apples, carrots, citrus fruits,
o Loop Diuretics: furosemide (Lasix); Bumetanide sweet potatoes
(Bumex) • One glass of wine a day - but skip this if you are at risk
• Monitor Daily weight and I&O - Pt should lose weight if for alcohol abuse or cancer
diuretic is effective Cholesterol Lowering Medications
• Most common side effect of diuretics is electrolyte • First cholesterol lower meds were bile sequestration
imbalance. Others - Postural hypotension, GI symptoms, medications - Cholestyramine (Questran) - had lots of
Sexual dysfunction side effects
• Potassium Sparing Diuretics - Inhibit aldosterone, cause • Newer and more popular meds fall into two basic
Na+ to be excreted in exchange for K+ categories:
o Spironolactone (Aldactone) - causes gynecomastia o Fibrates (including gemfibrozil [Lopid] and clofibrate)
Beta Blockers o Statins (such as lovastatin and provastatin).
• Atenolol (Tenormin); Metoprolol (Lopressor); Propranolol Bile Sequestrant Drugs
(Inderal); Nadolol (Corgard) • Cause excretion of bile acids, which makes the liver
• Should not be used in patient's with asthma, COPD, CHF manufacturer more bile acids using up more cholesterol.
and heart block • Cholestyramine (Questran) and colestipol (Colestid)
• Side Effects - Bronchospasm, Bradycardia, fatigue, sexual o Side effects - abdominal pain, N/V and bloating.
dysfunction, Peripheral vascular insufficiency • Naicin (nicotinic acid) limits conversion of lipids to LDL in
Alpha-1 Blockers the liver and causes increased production of bile acids
• Prazosin (Minipress); Terazosin (Hytrin) o Must be taken in large dosages
• Reduce peripheral resistance by dilating arterioles and o Common Side Effects -hot flashes and itchy, dry skin.
venules o Severe Side effects - Chemical hepatitis", tendency
• Side effects- syncope, orthostatic hypotension, weakness; toward diabetes, gout with arthritis and kidney
palpitations damage.
• Need to avoid standing for long periods of time Fibrates (Fibric Acid Derivatives)
• Need to rise slowly to avoid orthostatic hypotension • Gemfibrozil (Lopid) - works by defeating the liver's ability
Centrally Acting Alpha Blockers to make very low density lipoproteins, precursors to LDL,
• Clonidine (Catapres); Methyldopa (Aldomet) Also raises good (HDL) cholesterol.
• Activate central receptors that suppress vasomotor and o Abdominal pain, diarrhea, nausea, and vomiting may
cardiac centers. Leads to decrease in peripheral occur.
resistance • Clofibrate (Atromid), has higher toxicity which limits its
• Side Effects - Drowsiness, fatigue, sexual dysfunction, use.
Orthostatic Hypotension o Nausea, Flu-like symptoms, Agranulocytosis
Vasodilators Statins (reductase inhibitors)
• Hydralazine (Apressoline) • Lovastatin (Mevacor), Simvastatin (Zocor), Pravastatin
• Dilate peripheral blood vessels by directly relaxing (Pravachol), Fluvastatin (Lescol)
vascular smooth muscle o Interfere with the liver's ability to make cholesterol.
• Usually used in combination with other meds, because o Cause abnormal liver function in about two percent
they lead to sodium and fluid retention and cause reflex of the patients. Small incidence of muscle
cardiac stimulation inflammation
• Side effects - Headache, dizziness, tachycardia, fatigue, o Fewer reports of side effects with Pravastatin
edema o Take with meals and avoid alcohol
Calcium Antagonists Circulatory Problems
• Amlodipine besylate (Norvasc); Diltiazem (Cardizem); • Chronic circulatory problems lead to the following
Verapamil (Calan, Isoptin) Nifedipine (Procardia) changes
• Inhibit influx of calcium into muscle cells which acts to o Dry skin
reduce arterial spasms and promote vasodilatation o Dry brittle hair
• Side Effects - dizziness, fatigue, nausea, headache, o Loss of body hair
edema o Thick brittle nails (particularly toe nails)
Renin-Angiotensin - Aldosterone System o Increased capillary fill times (over 2 seconds)
o Capillary filling is a indicator of peripheral circulatory • May occur in both legs and often continues to worsen
status over time.
• Some people complain only of weakness in the legs when
Peripheral Vascular Disease (PVD) walking or a feeling of "tiredness" in the buttocks.
• characterized by lack of blood flow and oxygen to tissues • Impotence is an occasional complaint in men.
• Usually occurs as a result of accumulated fatty deposits • Pain at rest is uncommon.
• Most prevalent in geriatric clients, however, prevention of Arterial PVD
PVD needs to start at birth • Extremity has decreased oxygenation, may be pale, cool
• Smoking makes PVD worse, by constricting the blood or cyanotic
vessels and increasing the force of flow (increased B/P) • Sharp pain, may increase with activity
• Can be divided into Arterial and Venous PVD • Arterial insufficiency ulcers are usually dry, pale, and may
Assessment of PVD have necrotic tissue
• Check peripheral pulses - know names and location of • To treat, need to increase blood flow to extremity
femoral, brachial, radial, popliteal, posterior tibial and o place foot lower than heart
dorsalis pedis o Avoid tight bandages and stockings
• Check below the site of suspected occlusion o Need meticulous foot care to avoid ulcers
• If can’t palpate a pulse, use a doppler Gangrene Secondary to Arterial Occlusive Disease
• Check capillary filling Medications for Arterial PVD
• Check warmth and color by comparing extremities to • Need to dilate peripheral arteries
each other • Pentoxifylline (Trental) is drug of choice for PVD
• Arterial PVD Venous PVD - • Take Trental with meals
Arterial vs Venous PVD • People who are sensitive to caffeine or theophyllin can’t
• Arterial take Trental
• Cool • Side effects Dizziness, nervousness, agitation
• pale extremity Surgical Treatment of Arterial Peripheral Disease -
• decreased pulses Bypass Graft
• sharp pains • Frequently used to treat blockages of larger artery
• pain increases with activity or elevation • Most common - Femoral-Popliteal Bypass Graft
• Dry ulcer formation with necrotic tissue • Post op - Keep leg fairly straight, don’t elevate, monitor
• Venous for bleeding, check peripheral pulses and skin
• Warm temperature, bedrest, calf-pumps to prevent clot
• Flushed formation
• Edematous Venous PVD
• Aching pain • In venous PVD, extremity has too much blood (can’t get
• pain increases when legs dependent back to the heart)
• Weeping venous ulcers • Extremity is warm, flushed, edematous
Arterial PVD • Aching pain, gets worse with standing
• Obstruction or constriction of arterial flow, usually in the • Need to facilitate blood return
leg o Elevate legs
• Intermittent claudication is an early sign of arterial PVD o Use teds or ace wraps
(pain in calf or leg brought on by exercise and relieved by • Venous stasis ulcers tend to be pink or red and produce
rest) lots of drainage
• Later signs are loss of hair on leg and dorsum of foot, Vericose Veins
muscle atrophy and arterial ulcers • Venous defect allows pooling, particularly in peripheral
Thromboangitis Obliterans (Buerger’s Disease) veins with little support
• Obstructive inflammatory process of the peripheral • Tend to run in families
arteries • Peripheral veins can be removed (stripped)
o Mostly male smokers 20-40 years old • Post op, elevate legs when sitting. Avoid long periods of
o More often in Semitic and Oriental standing. Keep elastic bandages tight and wrinkle free,
o Causes Intermittent claudication, numbness and monitor for bleeding, check pedal pulses and edema
tingling, thrombophlebitis Deep Vein Thrombophlebitis (DVT)
o May require amputation of affected digits • Slowed blood flow allows a clot to form.
Raynaud’s Phenomenon • May have altered clotting mechanisms
• Episodes of arterial spasm - usually in hands • Patients on bedrest have higher risk of DVT
• primarily in women 20-40 • People who wear tight knee socks or have pillows under
• Cold, numbness, paleness, and pain in one or more knee (compresses popliteal) have increased risk of DVT
fingers or toes • To prevent DVT while on bedrest - do calf pumps
• Bilateral effects both hands and/or feet (dorsiflex and extend feet frequently)
• Smoking and Cold makes it worse • S/S of DVT - warm, red, edematous leg
• Intense redness and throbbing after spasm • May have positive Homan’s sign
• Tx with calcium antagonists, may require amputation • DVT can be silent, may be life threatening, and can lead
Arterial Embolism to embolism
• Blood clots (usual from heart) tend to lodge in bifurcation Treatment of DVT
of an artery, blocking blood flow • Bedrest may be needed during acute phase
• Symptoms depend on size and location • Avoid standing still and sitting
o Abrupt onset of pain, burning, loss of distal pulses, • Check pulse distal to site of thrombosis
cold pale extremity • Assess edema, measure calf circumference
• Tx with bedrest, and anticoagulants or fibrinolytics • Don’t massage leg
o Surgery - embolectomy or endarterectomy within 6- • Elevate leg on pillows, use warm moist heat
10 hours • Ted hose or other compression
Intermittent Claudication • Watch for signs of pulmonary embolism
• Due to narrowing or blockage of the arteries • NSAIDs for pain and inflammation
(Atherosclerosis or arteriosclerosis Obliterans - peripheral Anticoagulant Therapy
vascular disease). • Heparin
• Frequently one of the first signs of cardiovascular disease o Give heparin with smallest needle possible in SUB-Q
• Pain in legs that occurs with exercise. Pain is relieved by tissue of abdomen. Don’t aspirate, don’t massage.
rest. Avoid 1 inch area around umbilicus
o Monitor PTT time • Provides evaluation of how the heart functions during
o With all anticoagulants, hematest stools for blood exercise/stress
• For long term treatment - Warfarin (Coumadin) orally • Patient with suspected exercised induced arrhythmia or
o Monitor PT time (coumadin interferes with chest pain is attached to a telemetry unit
Prothrombin formation) Watch for bruising • Patient is placed on a treadmill and walks uphill until
Surgical Treatment of DVT chest pain/arrhythmia's are reproduced or maximal
• Large clots can be surgically removed desired cardiac output is reached (calculated for each
• For patient with high risk of repeated DVT, a Greenfield person)
vena cava filter can be used • Monitored by EKG the entire time
• Filter catches clots before they can migrate to the lungs, • Heart rate and B/P go up and lead to increased cardiac
heart or brain output without pain in healthy individual
• Used in conjuction with heparin or coumadin therapy Lab values
Duplex Ultrasound/Ultrasonography • Triglycerides - overall fat content of serum
• Examines the blood flow in the major arteries and veins in • Cholesterol - overall cholesterol level - Goal is to keep it
the arms and legs with the use of ultrasound and at or below 200
ultrasonography, which provides a visual image. o HDL High density lipoproteins - “good” cholesterol,
• Done in the ultrasound or radiology department. may act to protect blood vessels from damage
• Veins: A water-soluble gel is placed on the transducer and o LDL Low density lipoprotein “bad” cholesterol, acts
placed over vein as a “glue” to clog vessels
• Arteries: Blood pressure cuffs will be put around the Nitroglycerin - Drug of Choice for Angina
thigh, calf, and ankle to examine the legs. • Relaxes the smooth muscle tissue of the vascular system
Ateriogram/Venogram • Acts to dilate coronary arteries
• Injection of radiopaque dye into the femoral artery or vein • Increases blood flow to cardiac tissue
to determine where and how severe obstructions are • Increases oxygenation of cardiac muscle
• Determine if patient has previous allergies, particularly to • Common Side Effects
procedure dyes, iodine, shellfish or strawberries o headache, hypotension, dizziness
• After procedure, keep leg straight, apply pressure Application of Nitroglycerin
dressing to site. Watch site for bleeding. Monitor pulses • Sublingual - for acute chest pain. May take one tab under
distal to site. tongue every 5 minutes until pain is relieved (up to 3
tabs). If not relieved, call MD
Coronary Artery Disease (CAD) • Patches (Nitropatch/Nitrodur) - Remove old patch, apply
• Coronary arteries narrowed or completely blocked by new one to any clean, intact, non-hairy part of the body
fatty plaques (atherosclerosis). • Paste - apply the correct amount of paste to the
• inadequate blood supply to the heart muscle supplied by manufactures paper, then apply paper “patch” to
that artery. patient’s skin as above
• Often results in death of the involved area Other Meds to treat Angina
• of heart muscle. This is called a myocardial infarction • Calcium Channel Blockers
("MI") or heart attack. o Nifedipine (Procardia) - dilates both coronary arteries and
Myocardial Ischemia peripheral arterioles (decreases B/P)
• Myocardial Ischemia - less severe reduction in blood o Can be swallowed whole, or puncture capsule with a
supply where the heart muscle receives just enough needle and give contents sub-lingual
blood to stay alive. o Take Calcium Channel Blockers on empty stomach
• Either ischemia (poor blood supply) or infarction (virtually • Beta Blockers
no blood supply) can cause cardiac symptoms such as o Atenolol (Tenormin) and Nadolol (Corgard) Acts to
chest pain or shortness of breath. suppress renin-angiotensin-aldosterone system
• Angina is the name given to chest, neck, jaw, back, o Take Beta-blockers with food
shoulder or arm pain caused by heart muscle that is • Dipyridamole (Persantine) - selectively dilates coronary
ischemic (not receiving enough blood supply). arteries (does not affect B/P)
Angina Pectoris Echocardiogram
• Sharp, diffuse chest pain in response to insufficient • Echocardiogram
oxygen to cardiac muscle o Ultrasound of heart
• Brought on by exertion, cold, stress, over-eating o can view structures and movement
• Usually relieved by rest o Similar to fetal ultrasounds, painless, need to use
• Caused by insufficient blood flow through the coronary transducer gel
arteries, which leads to decreased oxygenation of cardiac o No real patient prep needed
tissue Transesophageal Echocardiography
• Deoxygenation of tissue leads to build up of lactic acid, • Uses probe to pass ultrasound transducer into the
and if continued, leads to ischemia esophagus
• Goal is to control Angina so patient can do ADL’s without • more detail is possible
chest pain • Keep pt NPO 4 hours before. Topical spray or gargle to
Diagnostic Tests depress gag reflex. Conscious sedation may be used.
• 12 Lead EKG or ECG • Keep pt NPO afterwards till gag reflex returns
o records electrical conduction of heart Coronary Angiograms /Veinograms
o Gives information about excitation of the • Coronary Angiography
myocardium o injection of radiopaque dye into the right and left
o Useful for detecting arrhymias coronary arteries
o Can detect evidence of MI o Helps to confirm the presence and /or degree of
o Telemetry - Three lead EKG used to monitor cardiac coronary artery stenosis, and to determine the
patients in bed amount of collateral circulation available
• Holter monitor • Ventriculography
o Similar to telemetry, but records results for 24 hours. o injection of dye into left ventricle to visualize
Can be used on outpatient basis, to detect ventricular wall, mitral valve and ventricular septum
arrthymia's which occur as a result of something the function
person does at home Cardiac Catheterization
Stress Test • Insertion of catheter directly into the right and/or left
• A number of stress tests are used, but the most common ventricle to get info about ventricular function
is the exercise stress test
• Gives most valuable and detailed information about • Uses radioactive tracer substances
structure and function of heart • Thallium imaging
o Patency of coronary arteries o Dye is injected into patient’s antecubital vein.
o Condition of myocardium o Imaging done 4-10 minutes after injection
o Status of collateral circulation o Necrotic or ischemic tissue will appear as “cold
• Relatively high risk compared to other cardiac tests spots” on this scan
Patient Preparation o Often done with the patient at rest and then during
• Explain expected sensations - flushing, nausea, warmth an exercise test
when dye injected Coronary Artery Bypass Graft (CABG)
• May have chest pain or dyspnea when catheter is in heart • CABG to relieve blockages of the coronary arteries
• Risks are hemorrhage, MI, CVA, dysrhythmia and even • Open Chest and long incision along the inside of the lower
death (most risk is from cardiac cath) leg.
• Pt is NPO 4 hours before, procedure lasts 1-3 hours, lying • The leg vein is sewn in above and below the blockage in
on x-ray table in cold room the coronary
• Post Cath, Pressure dressing to site, monitor for bleeding, • Heart-lung bypass machine is used to re-route the blood
Check peripheral pulses below site from the heart while the surgery is being done
PTCA Percutaneous Transluminal Coronary Angioplasty Indications for CABG
• In PTCA, a catheter carrying a small balloon is inserted • Unsuccessful Angioplasty
into coronary heart arteries through the patient's skin. • Blockage of the left main coronary artery
• The balloon is then repeatedly inflated and deflated to • > 50% diameter stenosis
compress the blockage that obstructs blood flow to the • Concomitant valve damage and any coronary artery
heart. disease
Post-Procedure Care • Unstable Angina and/or ischemia that cannot be treated
• Always assess for adequate peripheral circulation below with PTCA
the site of arterial access • Three-vessel disease of the coronary arteries regardless
• Assess site for bleeding, a pressure dressing will be in of angina or ischemia
place for at least 8 hours. CHF
• Keep affected extremity straight and still • Right Sided Failure - Complication of COPD
• Elevate HOB no more than 90 degrees o Pitting peripheral Edema
• Push fluids to get rid of dye (nephrotoxic) o Liver enlargement and right upper quadrant pain
• Assess heart rate and B/P o Ascities
• Monitor I&O for 24 hours o Distended neck veins (JVD)
Myocardial Infarction (MI) • Left Sided Failure - Complication of Hypertension
• Usually caused by the sudden occlusion of a coronary o Dyspnea - early sign
artery. o Cough
• The plaque in the artery takes years or decades to form, o Orthopnea
but the final event of formation of a blood clot (or o S 3 heart sound
thrombus) happens in a few minutes or hours. o Paroxysmal nocturnal dyspnea (PND)
• In general MIs occur at any time, although they are a bit o Fatigue
more common in the morning o Crackles in lungs - starts at bases and works upward
Symptoms of MI Congestive Heart Failure (CHF)
• Classical symptoms of a heart attack include heavy or • Most common hospital discharge diagnosis in persons
crushing chest pain, shortness of breath (dyspnea) and over age 65.
sweating (diaphoresis). • The National Heart, Lung and Blood Institute estimates
• Less common are nausea, profound fatigue and a feeling two million Americans are afflicted with CHF and 400,000
of impending doom. new cases of CHF are diagnosed every year.
• If you become short of breath and break out in a cold CHF
sweat while sitting in a cool room you should be more • CHF usually starts with L sided failure, S/S of pulmonary
concerned than if these symptoms happen while walking congestion and pulmonary edema
up a steep hill in July. • Chronic CHF leads to ventricular dilation, hypertrophy and
• Many heart attacks come without classical symptoms or tachycardia
even without any symptoms at all (silent MI). • Heart becomes enlarged and weaker
• Ischemic myocardium is electrically unstable • PMI may be located at 6th intercostal space and lateral to
o arrhythmia's including lethal ventricular ventricular the MCL
fibrillation and ventricular tachycardia. • Clubbing of fingers from chronic hypoxia
o Infarcts which interfere with the cardiac conduction Treatment of CHF
system may lead to heart block or conduction • Need to decrease fluid volume and reduce cardiac
defects. workload
• Dead or scarred myocardium is unable to contract. o Bedrest or reduced activity to conserve energy
• Damage may be reversible if thrombolytic therapy is in o Oxygen if hypoxia is present
time. o Assist to cough up secretions q 2 hours
• Leads to congestive heart failure in about 40-50% of o Elevate HOB, Do not raise feet, even if edematous
patients with acute MI. o Diuretics
• In extreme cases, patients may present with cardiogenic o Digitalis
shock o Fluid restrictions
Cardiac Isoenzymes o Daily weight
• Creatinine Kinase (CK) o I&O
o Normals < 2.5U F, < 4.3U Male o CHF Diet
o Elevations indicate possible brain, heart, or muscle CHF Diet -Low Cholesterol, Low Sodium, Low Fat Diet, -
injury or necrosis To Return B/P to Normal
• CK-MB (0-5% of total CK) • Avoid the following foods
o Elevations with myocardial injury • Red meats, Cheese (except cottage cheese and other low
• Lactic Dehydrogenase (LDH) fat), Egg yolks, Shellfish (too much cholesterol)
o LDH1/LDH2 LDH 1 higher than LDH2 with myocardial • Cold cuts, ham, bacon, sausage, hot-dogs, chips, (sodium
damage and fat)
o Most sensitive cardiac enzyme indicator of MI • Most soups, sodas, and canned vegetables (sodium)
Nuclear Imaging • Antacids and other meds high in sodium
Alternatives
• Use lite salt (K+Cl)
• Use salt alternatives - Mrs. Dash and others
• Bake and season meats with vinegar or lemon juice
instead of butter and salt
• Use egg beaters, low sodium soups, saltless pretzels and
low sodium crackers.
• Try a baked sweet potato with a little brown sugar
• Read all labels
Low Calorie
• Watch for “Hidden” calories in drinks (milk, juice, alcohol,
soda)
• Watch the fat, sodium content and calorie content in
frozen dinners, mixes, and canned items
• Avoid High carbohydrate foods (breads, pastas, cakes)
• Avoid most fast foods
• Read the labels. Check out nutritional content
Digoxin (Lanoxin) - Cardiac Glycoside
• Cardiac Glycosides act to stimulate myocardial
contraction
• Makes heart beat slower, more regular, and more
powerful, which leads to increased cardiac output and
increased blood flow to the kidneys
• Can be given PO or IV
• Take Apical pulse for a full minute before administration,
noting rate and rhythm.
• If rate is below 60, above 110, or If rhythm is very
irregular (compared to patient’s baseline, hold med and
call MD
• Dig has a narrow therapeutic window -Watch serum
levels (0.8-2.0 ng/ml)
• Don’t take with antacids - interfere with absorption
• Persons with hypokalemia are more likely to become
digtoxic so patient needs to eat foods high in potassium
(know what these are)
• Patients on Lasix and Dig - watch for toxicity
• Antibiotics and/or calcium channel blockers can interact
with Digoxin causing increased risk for toxicity
• Digitalization - when first starting on Dig, given higher
dose - called a loading dose or a digitizing dose
• After patient responds, dose is lowered to a Maintenance
dose
• Maintenance dose is one which allows heart to function
without toxic symptoms
Signs and Symptoms of Digitalis Toxicity (serum levels
above 2.0 ng/ml)
• Cardiovascular
o Bradycardia or Tachycardia
o Bigeminy
o Ectopic beats
o Pulse deficit
• GI Effects*
o N/V
o Anorexia
o Diarrhea
o Abdominal Pain
• Neurological Effects
o Headache
o * Blurred, double or colored vision
o Confusion, irritability, restlessness, drowsiness
o Muscle weakness and cramps
• Kidney Failure, Liver Failure, Potassium Loss or Dig
Overdose can all cause Dig Toxicity
Rotating Tourniquets
• old technique used to decrease venous return to the
heart by trapping fluid in extremities
• temporarily reduces heart workload by reducing fluid
volume heart must pump around body
• B/P cuffs are used on three extremities at a time, and are
rotated every 15 minutes in a clockwise fashion.
(Rotating tourniquet machine does this automatically)
• Make sure patient has a peripheral pulse in affected
areas

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