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Otitis Media With Effusion: Presentation

The document discusses various ear conditions including otitis media with effusion, acute otitis media in children, acoustic neuroma, Meniere's disease, otosclerosis, peritonsillar abscess, tonsillar carcinoma, and hearing screening tests in children. Diagnosis involves examination of the ear canal and eardrum, audiometry, tympanometry, and MRI imaging. Treatments include antibiotics, surgery such as grommet insertion or stapedectomy, and hearing aids.

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0% found this document useful (0 votes)
27 views

Otitis Media With Effusion: Presentation

The document discusses various ear conditions including otitis media with effusion, acute otitis media in children, acoustic neuroma, Meniere's disease, otosclerosis, peritonsillar abscess, tonsillar carcinoma, and hearing screening tests in children. Diagnosis involves examination of the ear canal and eardrum, audiometry, tympanometry, and MRI imaging. Treatments include antibiotics, surgery such as grommet insertion or stapedectomy, and hearing aids.

Uploaded by

Wonjoo Lee
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ENT

Otitis media with effusion


➢ Also known as glue ear, persistent build-up of fluid in the middle ear
➢ History of recurrent OM
➢ Issue stems from a dysfunction of the Eustachian tubes
Presentation
• Hearing diminished or lost (usually bilateral) → glue ear is the commonest cause of CHL in childhood
- Listening to the TV at excessively high volumes
- Needs things to be repeated
- Lack of concentration
- Withdrawal especially in school
• 2ry problems such as speech and language delay or behavioral problems
• Rarely complains of ear pain
• May have a prior history of infections (URTIs) or oversized adenoids
• Balance problems
• Peaks at 2 years of age
Signs (by otoscope)
• Retracted (more common) or bulging (less common) drum
• Dull, grey, or yellow tympanic membrane, there may be bubbles or a fluid level
Diagnosis
• Audiograms → conductive defects
• Tympanometry → assesses the ability of the eardrum to react to sound
Treatment
• <3 months → Reassure and review in 3 months
• >3 months → Surgery (insert grommets)
• If surgery is rejected → Hearing aids

- An important risk factor for OME is “parental smoking” → Always encourage parents of patients to stop smoking

- The light reflex (cone of light) is seen as a cone-shaped reflection in the anterior inferior quadrant of the TM
- Absence of the cone of light indicates distortion of the shape of the TM such as bulging due to an increase of
inner ear pressure seen in otitis media

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Acute otitis media in children
➢ Acute inflammation of the middle ear and may be caused by bacteria or viruses
Features
• Rapid onset of pain (younger children may pull at the ear)
• Fever
Treatment of perforated OM
• Irritability
• Amoxicillin (5-days course)
• Coryza (rhinitis) • If penicillin-allergic → Erythromycin or clarithromycin
• Vomiting
• Often after a viral upper respiratory infection
• A red, yellow or cloudy tympanic membrane or bulging of the tympanic membrane
• An air-fluid level behind the tympanic membrane
• Discharge in the auditory canal secondary to perforation of the tympanic membrane
• Perforation of the eardrum often relieves pain. This is because bulging of the tympanic membrane causes
the pain

• Furuncles can be
found in diabetics or
low immunity
• Also called “boils”
• They’re infected hair
follicles
• MC organism →
Staph
• Red, hard, tender
• Self-limiting or
requires flucloxacillin

- Tenderness in movement of the tragus → Otitis externa


- If you’re treating otitis externa but suspect there may be a tympanic membrane perforation → aminoglycosides
ear drops are NOT the best choice as it’s toxic → use Ciprofloxacin drops

- Otitis externa with Pseudomonas (pus in the external canal) → topical gentamicin only or with topical
gentamicin with hydrocortisone (Gentisate HC)
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Acoustic neuroma
➢ Also called “vestibular schwannomas”, accounts for 5% of intracranial tumors and 90% of cerebellopontine
angle
➢ Bilateral acoustic neuromas are seen in neurofibromatosis type II
Features
• Cranial nerve V → Absent corneal reflex
• Unilateral SNHL should be considered as caused by an
• Cranial nerve VII → Facial palsy acoustic neuroma until proven otherwise
• Cranial nerve VIII → SNHL, vertigo, tinnitus
Investigation
• MRI of the internal auditory meatus → to view the cerebellopontine angle
• MRI brain → for further evaluation

DVT = Deafness, Vertigo, Tinnitus


DVT + CN palsy = Acoustic neuroma
DVT + aural heaviness = Meniere’s disease

Meniere’s disease
Presentation
• Deafness, vertigo, tinnitus (DVT) + fullness in the ear (could be experienced with AN)
• Note: Vertigo → is usually the prominent symptom
• Episodes last minutes to hours
• MRI is normal
• Usually a female → male; 20-60 years old
• Typically, symptoms are unilateral but bilateral symptoms may develop after a number of years
Treatment
• Acute attacks → buccal or intramuscular prochlorperazine or cyclizine
• Admission is sometimes required

Vestibular schwannoma → high frequency SNHL, MRI is diagnostic, will have CN involvement
Meniere’s disease → low frequency SNHL
Otosclerosis → CHL + young age + patient reports better hearing in noisy places + precipitating factors like pregnancy

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Female Nurses and Doctors
ENT
Otosclerosis
• Abnormal growth of bones of the middle ear preventing structures within the ear from working properly
• Most common cause of progressive deafness in young adults (CHL) (15-45 years)
• Tinnitus and vertigo
• Positive family history (50%) • Could be mixed hearing loss if cochlea is involved
• Usually bilateral (80%)
• Women + pregnant
Management
• Stapedectomy or stapedotomy, with the insertion of a prosthesis
• If surgery is rejected → Hearing aids

Peritonsillar abscess (Quinsy)


➢ Swelling of the soft palate and tissues lateral to the tonsils
➢ It’s a complication of acute tonsillitis
➢ Pus is trapped between the tonsillar capsule and the lateral pharyngeal wall
➢ Typically preceded by a sore throat for several days
Presentation
• Sore throat
• Dysphagia
• Pain localized to one side of the throat
• Peritonsillar bulge
• Uvular deviation (bulging tonsils push the uvula away from the affected side)
• Fever
• Trismus (difficulty opening the mouth)
• Fetor
• Drooling
• Altered voice quality (hot potato voice) due to pharyngeal edema and trismus
Management
• Antibiotics → IV benzylpenicillin
• Needle aspiration, incision and drainage

Tonsillar carcinoma → SCC, Lump in the neck


- RF: smoking, regular alcohol intake and HPV infection
- Direct spread often involves pharyngeal space and mandible
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ENT
Hearing screening
➢ It’s offered to all babies within 4-5 weeks of birth
➢ Healthy born babies are normally given a hearing test before discharge otherwise it’ll be done during this
timeframe
2 types of tests
1. Automated otoacoustic emission (AOE)
- Soft-tipped earpiece is placed inside the ear to detect vibration of hair cells
2. Automated auditory brainstem response (AABR)
- Brain waves are measured by electrodes
- Can detect auditory neuropathy in children

Below 6 months → AOE or AABR


6 months – 18 months → Distraction test
2 years – 5 years → Conditioned response audiometry OR Speech discrimination
>5 years → Pure tone audiogram

Rinne’s and Weber’s

Rinne’s test
• A tuning fork is placed over the mastoid process until the sound is no longer heard, followed by repositioning
just over external acoustic meatus
• AC is normally better than BC → Positive Rinne's test (normal)
• If BC > AC, then the patient has conductive deafness → Negative Rinne’s test (Abnormal Rinne’s test)

Weber's test [CSSO]


• A tuning fork is placed in the middle of the forehead equidistant from the patient's ears. The patient is then
asked which side is loudest
• If sound is localized to the unaffected side → Unilateral SNHL
• If sound is localized to the affected side → Unilateral CHL
• SNHL = localized to the normal ear

Example
If Weber’s test localizes to the right side. It can either be right CHL or left SNHL, a Rinne’s test would be able to
confirm if it’s a right CHL

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Candida vs LP vs Leukoplakia

Leukoplakia should be biopsied as it’s premalignant


Leukoplakia occurs primarily in HIV-positive individuals
For oral candidiasis → Nystatin suspension can be used
Leukoplakia may present with dysplasia of the vocal cords
- White patches over the vocal cord + hoarseness of voice
- Management → cessation of smoking and observation, as it might turn malignant
Difference between oral thrush (oral candidiasis) and Leukoplakia → Leukoplakia CANNOT be removed by rubbing
Ludwig’s angina
- Severe cellulitis involving the floor of the mouth
- Early, the floor of the mouth is raised and there’s difficulty swallowing saliva which might be drooling
- Usually following a dental infection
Dysphagia + Odynophagia + radiates to the back → Esophageal candidiasis, caused by candida albicans

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Cholesteatoma
➢ Destructive and expanding growth consisting of keratinizing squamous epithelium in the middle ear and/or
mastoid process
➢ Uncommon abnormal collection of skin in the ear that left untreated can continue to grow and damage the
bones of the middle ear (ossicles)
➢ Small lesions → CHL, Large lesions → CHL + vertigo + headache + facial nerve palsy
Acquired
• Following repeated ear infections, they’re usually responsive to antibiotics
• Frequent painless otorrhea which may be foul-smelling
• Progressive, unilateral CHL
• TM perforation (90%) or retracted tympanum
• Otoscopy:
- Retraction pocket in attic or posterosuperior quadrant of TM
- Granular tissue
- White mass behind eardrum
- Purulent drainage
Congenital
• Present 6 months – 5 years, sometimes later in adulthood
• Often NO history of recurrent suppurative ear disease, previous ear surgery or TM perforation
• May be incidental finding on routine otoscopy of an asymptomatic child
• Otoscopy → Spherical pearly white mass behind intact membrane

Vestibular neuritis
➢ Inflammation of the vestibulocochlear nerve (CN VIII), but the etiology is thought to be a vestibular
neuropathy
Features
• Abrupt onset
• Recurrent vertigo (lasting hours-days)
• Unsteadiness, nausea and vomiting (feel as if the room is rotating)
• Symptoms are aggravated by head movement
• History of viral infection (runny nose, cough, fever)

Vestibular neuritis → commonly present with a history of viral infection + lasts hours-days
BPPV → lasts seconds
Labyrinthitis
- Vestibular nerve and labyrinth are affected
- Same as vestibular neuritis + Hearing loss (SNHL) ± tinnitus
Vertebrobasilar insufficiency (VBI)
- Very old male
- Most common cause → atherosclerosis
- RF: DM, HTN, smoking and dyslipidemia

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Temporomandibular disorders (TMDs)
• A group of disorders affecting tempomandibular joint (TMJ), masticatory muscles and associated structures
• Associated with muscle overactivity which include bruxism (grinding of teeth)
• Symptoms → Facial pain, restricted jaw function and joint noise
• Pain is around the temporomandibular joint but is often referred to the head, neck and ear
• Managed by ice packs, NSAIDs, dental splits or Botox injections

Pleomorphic adenoma
• The most common tumor of the parotid gland
• They’re benign tumors which appear as a lump just behind the angle of mandible
• Benign with the capacity to turn malignant
Features
• Slow-growing and asymptomatic
• Firm
• Painless
• Mobile
Management
• Superficial parotidectomy or enucleation
- Tender and painful mass at the angle of the mandible, especially when eating → Parotiditis
- Mobile, soft, cystic and tender mass → Adenolymphoma (Warthin’s tumor)
- Mandibular and tonsillar tumors are NOT mobile

Sialadenitis
➢ Inflammation of salivary gland and may be acute or chronic, infective or autoimmune
Features
• Unilateral redness, swelling and pain
• May enlarge to reach a size of an orange
• Mild odynophagia, usually common before and during meals
• Fluctuation test positive if it’s filled with swelling
• Foul taste in the mouth
• Decreased mobility in the jaw
• Dry mouth, skin changes, weight loss, shortness of breath, keratitis, dental pain, skin changes and
lymphadenopathy
• Fever with rigors and chills along with malaise and generalized weakness as a result of septicemia
• In severe cases → pus can often be secreted from the duct by compressing the affected gland
• Duct orifice is reddened with reduced flow, there may be a visible or palpable stone
Acute sialadenitis
- Typically, present with erythema over the area, pain, tenderness on palpation and swelling
- Infection often occurs as a result of dehydration with overgrowth of oral flora (e.g. postoperative
dehydration)
- Purulent material may be observed
Chronic sialadenitis
- Less painful + gland enlargement (often following meals) WITHOUT erythema
- Associated with decreased salivary flow due to stones rather than dehydration

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Nasal polyps
• Lesions arising from the nasal mucosa, occurring at any site in the nasal cavity or paranasal sinuses
• Nasal polyps tend to be bilateral
• Associated with → Asthma, Aspirin sensitivity (Samter’s triad)
Presentation [RAN]
• Rhinorrhea
• Anosmia (loss of smell)
• Nasal obstruction

Laryngeal cancer
Presentation
• Progressive hoarseness of voice → most common early symptom
• Later, Stridor, dysphagia and odynophagia
• If the pharynx is involved → Hemoptysis and ear pain • HPV → RF for tonsillar, oropharyngeal and
Risk factors laryngeal cancer
• Smoking → 1 st

• Occupational exposures (asbestos, formaldehyde, nickel, isopropyl alcohol and sulphuric acid mist)
• Insufficient fruit and vegetables intake
• HPV 16

Nasopharyngeal carcinoma
• Painless swelling or lump in the upper neck, often due to a swollen LN
• Nasal obstruction, epistaxis and otitis media from eustachian tube obstruction
• Unilateral CHL + tinnitus
• Other cranial nerves involvements
Risk factors EBV is associated with:
• Smoking, Alcohol • Hodgkin’s lymphoma
• Infection with EBV • Nasopharyngeal carcinoma

• South Asian, male

Nasopharyngeal carcinoma → Ear symptoms


Paranasal sinus tumors → Prominent ocular symptoms (e.g. epiphora, double vision)
Oropharyngeal cancer (tonsillar cancer)
- At the base of tongue, tonsils, soft palate and walls of the pharynx
- Associated with ear pain
- Often involves pharyngeal space and mandible
Malignant otitis externa
- An aggressive infection rather than a malignancy
- CHL + foul-smelling purulent otorrhea + facial nerve palsy
- Black skin around the ear
- RF: DM, weakened immune system
- Urgent refer to an ENT specialist is usually needed

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Acute tonsillitis
Symptoms
• Sore throat
• Pain referred to the ear
Signs
• Throat is reddened
• Tonsils are swollen and may be coated or have white flecks of pus on them
• Fever
• Swollen regional LNs
• Examination shows intense erythema of tonsils and pharynx, yellow exudate and tender, enlarged anterior
cervical glands

3 or 4 of the Centor Criteria → Bacterial tonsillitis → Antibiotics (Penicillin V)


• Fever >38
• Tonsillar exudates
• No cough
• Tender anterior cervical
lymphadenopathy

Infectious mononucleosis
- Affects teenagers more often
- Very large purulent tonsils and long-
lasting lethargy
- Splenomegaly

Tonsillectomy
• Aim → Reducing the incidence if recurrent infections
• Indications → children with no other explanation for recurrent symptoms + frequency of symptoms
- >7 episodes per year for one year
- >5 episodes for 2 years
- >3 episodes for 3 years
• Complications
- Primary hemorrhage → may require a return to theatre
- Secondary hemorrhage → antibiotics and antiseptic mouthwashes

Otherwise, tonsillitis is treated with Analgesics (paracetamol and ibuprofen)


If tonsillitis + exudates + cervical lymphadenopathy + fever >38 → Antibiotics

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Presbycusis
➢ Also known as age-related SNHL → most common cause of hearing impairment in elderly patients
➢ Etiology → degenerative changes in the inner ear (hair cells inside the cochlea)
Features
• Progressive high-frequency hearing loss
• Bilateral
• Usually occurs after age 50
• Difficulty understanding speech, especially in noisy environments
• Usually brought in by the family to clinics as the patient would not think his/her hearing is impaired
Management
• Hearing aids, to increase the high-frequency sound

The graph shows that hearing threshold level goes down as the frequency increases which is a feature seen in
presbycusis

Functional dysphonia
➢ Disturbance of voice in the absence of any structural abnormality of the larynx or any cord paralysis
➢ Diagnosed by exclusion
➢ There may be various interacting causes such as overuse of the voice, poor vocal technique and stress
➢ May occur after treatment of acute respiratory infection (if prior ttt of infection → Laryngitis)
Features
• Vocal fatigue (voice becoming worse with use) and laryngeal discomfort

Noise-induced hearing loss (NIHL)


• Hearing impairment resulting from exposure to loud sound
• Usually bilateral high frequency SNLH

- Acoustic trauma such as gun shooting or bomb explosion and barotrauma (such as sudden changes in pressure
like driving) → TM perforation → CHL
- Acoustic trauma → Sudden
- NIHL → Chronic

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Management of ear wax buildup
1. Ear wax softeners
- Sodium bicarbonate, sodium chloride, olive oil can be used
- Prescribe for 2-3 days initially
2. If symptoms persist → Ear irrigation
3. If irrigation is unsuccessful
- Ear drops are advised for further 3-4 days and then return for further irrigation
- Instill water into the ear, after 15 mins → irrigate the ear
- Refer to ENT specialist

Management of epistaxis
1. Lean forward, open mouth, press soft nose for 10-15 minutes
2. Nasal cautery with silver nitrate
3. Nasal packing

After stopping the bleeding, if unstable → transfer to A&E

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Notes
• An insect buzzing and stuck in the external ear → 2% Lidocaine, to kill it then remove it by Olive oil
• Soft objects, organic matter or seeds stuck in the ear → Suction with a small catheter
• Large clearly visible foreign bodies in adults or older children → Bayonet forceps
• Styrofoam or chewing gum → Acetone
• Superglue → Manual removal, in 1-2days after desquamation, or referral to the ENT specialist
• Batteries → Urgent ENT referral, removed within 24h
• Ear wax → Olive oil, to loosen the hard wax
• Any spherical object → Hook, can’t be grasped by forceps
• An intellectually-disabled patient with a foreign object in the ear → Removal under general anesthesia
• Indications for ENT referral:
- Uncooperative patient
- Requiring sedation
- Perforated ear drum
- An adhesive in contact with the eardrum
- Difficulty removing the foreign body
• Ear trauma with bleeding, tinnitus and CHL, possible nausea and vomiting initial investigation → Otoscopy
• RTA with bleeding and CSF leakage from the ear (a possible basilar fracture) → CT scan
• Small perforation the TM → Reassure
• Large perforation of the TM → Refer to a specialist
• Flamingo pink (Schwartz sign) → Otosclerosis
• Cartwheel appearance of the TM → Acute suppurative otitis media
• Chalky white patches on the TM → Tympanosclerosis
• Sudden vertigo + vomiting + preceding URTI → Labyrinthitis
• Difficulty hearing in noisy environment → Presbycusis
• Difficulty hearing in quiet environment → Otosclerosis
• Form of acute OM where vesicles develop on the TM, pain occurs suddenly and persists for 24h-48h, hearing
loss and fever suggest a bacterial origin → Myringitis
• Any salivary gland mass for more than 1 month → FNAC
• Paget’s disease + OI → Mixed hearing loss
• Headache worsens when bending forward + NO nausea or vomiting → Chronic sinusitis
• Headache worsens when bending forward + nausea, vomiting, photosensitivity → Migraine
• Swelling that moves up on swallowing → Goiter
• Moves up on swallowing and tongue protrusion → Thyroglossal cyst
• Fluctuant and transluminate → Cystic hygroma, a fluid-filled sac due to a blockage in the lymphatic system
• Pain at the cheeks preceded by URTIs + upper jaw pain/toothache → Maxillary sinusitis
• Same but pain at the nose bridge → Ethmoidal sinusitis
• Same but pain above the orbit → Frontal sinusitis
• Causes of SNHL:
- Acoustic neuroma
- Meniere’s disease
- Presbycusis → Bilateral
- Labyrinthitis
• Blunt trauma to the ear pinna with only redness → Oral analgesia
• Blunt trauma to the pinna with hematoma (bluish discoloration) → Incision and drainage + Oral antibiotics
• If left untreated, it will lead to → Cauliflower ear
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