Periodontology Assignment
Periodontology Assignment
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Periodontitis
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attachment and bone loss. Physiological bone levels range from 1.0 to
3.0 mm apical to the cemento-enamel junction. In addition, there is no
probing attachment loss.
Gingivitis:
Gingivitis is a non-destructive type of periodontal disease which is an often
painful inflammation of the gingiva. Gingivitis most commonly occurs due to bacterial
buildup on the teeth. The main symptom of gingivitis is red, puffy gingiva, bleeding
during brushes of teeth. This problem can be resolves with brushing, and regular
flossing as well as with antiseptic mouthwash may help. Gingivitis can progress to
periodontitis if a person does not treat it properly.
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Diagnostic look-up table for gingival health or dental plaque-induced gingivitis in
clinical practice
▪ Genetic/developmental
▪ Infections
▪ conditions Inflammatory/immune
▪ Reactive processes
▪ Neoplasms
▪ Endocrine/metabolic diseases
▪ Traumatic lesions
▪ Gingival pigmentation
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2017 classification of periodontitis, staging, and grading :
Periodontitis
Associated
with Systemic
Necrotizing Diseases
Periodontal Periodontitis
Diseases
periodontitis
Patients in this condition have clinical features may include necrotic papillary and
marginal gingiva, bleeding, pain, and marginal gingiva that is covered by a
yellowish white slough or pseudomembrane. Alcohol misuse play important in the
predisposition of necrotizing periodontal diseases as well as affects immune
responses. Severe necrotizing gingivitis can lead to necrosis of the alveolar bone,
resulting in necrotizing stomatitis in the immunocompromised individuals. If left
untreated, necrotizing stomatitis results in extensive osteonecrosis and, in some
extreme cases, noma. Three forms of necrotizing periodontal diseases have been
described in 2017:
► Necrotizing gingivitis
► Necrotizing periodontitis
► Necrotizing stomatitis and noma (cancrum oris)
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3- Periodontitis: inflammation of the supporting tissues of the teeth which
caused by specific or groups microorganism, that resulting in progressive
destruction of the periodontal ligament and alveolar bone with increased
probing depth formation, recession, or both. Clinically periodontitis can be
distinguished from gingivitis via detectable attachment loss, formation of
periodontal pocket, change in the density and height of the alveolar bone.
Changes in color, contour, and consistency, as well as bleeding with probing all
of them are clinical signs of periodontitis. In periodontitis, attachment loss
occurs either continuously or as in episodic burst of disease activity.
o Stage I: located between gingivitis and periodontitis, shows the early attachment
loss. Persistence of gingival inflammation and biofilm dysbiosis lead to stage I
periodontitis. Clinical attachment loss at a relatively early age may have
heightened susceptibility to disease onset.
o Stage II: At this stage of the disease process that represents established
periodontitis in which a carefully performed clinical periodontal examination
identifies the characteristic damages that periodontitis has caused to tooth
support. management of stage II relatively simple for many cases as application
of standard treatment principles involving regular personal and professional
bacterial removal and monitoring is expected to arrest disease progression.
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o Stage III: significant damage to the attachment apparatus and, in the absence of
advanced treatment, tooth loss may occur. presence of deep periodontal lesions
that extend to the middle portion of the root and whose management is
complicated by the presence of deep intrabony defects, furcation involvement,
history of periodontal tooth loss/exfoliation, and presence of localized ridge
defects that complicate implant tooth replacement.
o Stage IV: considerable damage to the periodontal support and may cause
significant tooth loss, and this translates to loss of masticatory function. In this
stage, the dentition is at risk of being lost, In the absence of proper control of the
periodontitis and adequate rehabilitation. In stage IV, deep periodontal lesions
that extend to the apical portion of the root and/or history of multiple tooth loss
as well as this stage complicated by tooth hypermobility because of secondary
occlusal trauma and the sequelae of tooth loss.
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Reference:
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