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• Binge eating - means consuming a large amount of food (far greater than
most
people eat at one time) in a discrete period of usually 2 hours or less.
• Purging
> involves compensatory behaviors designed to eliminate food by means of:
-self-induced vomiting or misuse of laxatives
-Enemas
-diuretics
Anorexia
• actually a misnomer
• clients do not lose their appetites.
• still experience hunger but ignore it and signs of physical weakness
and fatigue
• often believe that if they eat anything, they will not be able to
stop eating and will become fat.
• often are preoccupied with food-related activities such as: grocery shopping,
collecting recipes or cookbooks counting calories, creating fat-free meals cooking
family meals.
• may engage in unusual or ritualistic food behaviors such as: refusing to eat
around others cutting food into minute pieces
not allowing the food they eat to touch their lips.
Bulimia nervosa
•an eating disorder characterized by recurrent episodes (at least twice a week for
3 months) of binge eating followed by inappropriate compensatory behaviors to avoid
weight gain such as:
Purging fasting,
•excessively exercising
•Binging or purging episodes are often precipitated by strong emotions and followed
by:
•guilt, remorse
•shame, or self-contempt
RELATED DISORDER
Binge eating disorder
• characterized by recurrent episodes of binge eating
• no regular use of inappropriate compensatory behaviors, such as
purging or excessive exercise or abuse of laxatives; guilt, shame, and disgust
about eating behaviors; and marked psychological distress.
• frequently affects people over age 35, and it occurs more often in
men than does any other eating disorder.
Orthorexia nervosa
•sometimes called orthorexia
• is an obsession with proper or healthful eating.
• not formally recognized in the Diagnostic and Statistical Manual of
Mental Disorders, fifth edition
• Others believe it is a type of anorexia or a form of obsessive-
compulsive disorder.
• Behaviors include:
• compulsive checking of ingredients
• cutting out increasing number of food groups
• inability to eat only "healthy" or "pure" foods
• unusual interest in what others eat
• hours spent thinking about food. what will be served at an event;
• obsessive involvement in food blogs (Costa, Hardi-Khalil, & Gibbs,
2017)
ETIOLOGY
• A specific cause for eating disorders is unknown.
> Initially, dieting may be the stimulus that leads to their development.
• Biologic vulnerability
• developmental problems
• family and social influences can turn dieting into an eating disorder
Biologic Factors
•Disruptions of the nuclei of the hypothalamus may produce many of the symptoms of
eating disorders.
⁃ Deficits in the lateral hypothalamus result in decreased eating and
decreased responses to sensory stimuli.
• Disruption of the ventromedial hypothalamus leads to:
• excessive eating v weight gain
decreased responsiveness to the satiety effects of glucose
ANOREXIA NERVOSA
Onset and Clinical Course
• typically begins between 14 and 18 years of age.
•clients often deny they have a negative body image or anxiety regarding their
appearance.
•very pleased with their ability to control their weight.
Medical Management
focuses on :
•weight restoration
nutritional rehabilitation
Rehydration
•correction of electrolyte imbalances
Psychopharmacology
•Several classes of drugs have been studied, but few have shown clinical success.
• Amitriptyline (Elavil)
• Cyproheptadine (Periactin) in high doses (up to 28 mg/day) can
promote weight gain in inpatients with anorexia nervosa.
• Olanzapine (Zyprexa)
Flouxetine (Prozac)
•has some effectiveness in preventing relapse in clients whose weight has been
partially or completely restored.
Psychotherapy
• Family therapy may be beneficial for families of clients younger than
18 years.
• Family therapy is useful to help members to be effective participants
in the client's treatment.
•In a dysfunctional family, significant improvements in family functioning may take
2 years or more.
• Cognitive- behavioral therapy (CT),
BULIMIA NERVOSA
Onset and Clinical Course
•Bulimia nervosa usually begins in late adolescence or early adulthood
•18 or 19 years is the typical age of onset.
• frequently begins during or after dieting.
• Between binging and purging episodes, clients may eat restrictively,
choosing salads and other low-calorie foods.
BULIMIA
• may be underweight or over - weight
•General appearance is not unusual, and they appear open and willing to talk.
NURSING INTERVENTIONS
For Eating Disorders
Establishing nutritional eating patterns
• Sit with the client during meals and snacks
• Offer liquid protein supplement if client is unable to complete meal.
• Adhere to treatment program guidelines regarding restrictions.
• Observe the client following meals and snacks for 1 to 2 hours.
• Weigh the client daily in uniform clothing.
• Be alert for attempts to hide or discard food or inflate weight.
NURSING INTERVENTIONS
For Eating Disorders
Establishing nutritional eating patterns
• Sit with the client during meals and snacks
• Offer liquid protein supplement if client is unable to complete meal.
• Adhere to treatment program guidelines regarding restrictions.
• Observe the client following meals and snacks for 1 to 2 hours.
• Weigh the client daily in uniform clothing.
• Be alert for attempts to hide or discard food or inflate weight.
Helping the client identify emotions and develop non-food-related coping strategies
• Ask the client to identify feelings.
• Self-monitoring using a journal
• Relaxation techniques Distraction
• Assist the client in changing stereotypical beliefs.
2. Conversion disorder
• sometimes called conversion reaction
> involves unexplained, usually sudden deficits in sensory or motor function
deficits suggest a neurologic disorder but are associated with psychologic factors.
• An attitude of la belle indifférence, a seeming lack of concern or distress, is a
key feature.
> is a French term that translates to "beautiful ignorance."
3. Pain disorder
- has the primary physical symptom of pain, which generally is unrelieved by
analgesics and greatly affected by psychologic factors in terms of:
onset
Severity :
Exacerbation maintenance
RELATED DISORDERS
Malingering
• the intentional production of false or grossly exaggerated physical
or psychologic symptoms.
• motivated by external incentives such as: avoiding work
• evading criminal prosecution obtaining financial compensation obtaining
drugs
• have no real physical symptoms or grossly exaggerate relatively minor
symptoms
• Their purpose: external incentive or outcome that they view as
important and results directly from the illness.
Factitious disorder
• occurs when a person intentionally produces or feigns physical or
psychologic symptoms solely to gain attention.
• may even inflict injury on themselves to receive attention
• common term for factitious disorder is Munchausen syndrome.
•also called fabricated or induced illness
Munchausen Syndrome
ETIOLOGY
Psychosocial Theories
• people with somatoform disorders keep stress, anxiety, or frustration
inside rather than expressing them outwardly internalization.
• Clients express these internalized feelings and stress through
physical symptoms (somatization).
Biologic Theories
• differences in the way that clients with somatoform disorders
regulate and interpret stimuli.
• cannot sort relevant from irrelevant stimuli and respond equally to
both types.
• they may experience a normal body sensation such as peristalsis and
attach a pathologic rather than a normal meaning to it (Hollifield, 2005)
• Too little inhibition of sensory input amplifies awareness of
physical symptoms and exaggerates response to bodily sensations
• For example, minor discomfort such as muscle tightness becomes
amplified because of the client's concern and attention to the tightness.
TREATMENT
• focuses on managing symptoms and improving quality of life.
• health care provider must show empathy and sensitivity to the
client's physical complaints.
• A trusting relationship helps to ensure that clients stay with and
receive care from one provider instead of "doctor shopping."
SERTRALINE (Zoloft)
50- 200 mg/day
Monitor for nausea, loss of appetite, dizziness, dry mouth, somnolence or insomnia,
sweating, sexual dysfunction; avoid alcohol
PAROXETINE (Paxil)
v 20- 60 mg/day
Monitor for nausea, loss of appetite, dizziness, dry mouth, somnolence or insomnia,
sexual dysfunction; avoid alcohol