Mental Health Changes and Characteristics
Mental Health Changes and Characteristics
OCD
Obsessive-Compulsive Disorder (OCD) is a mental health condition
characterized by two main components:
Obsessions: Repeated, intrusive, and unwanted thoughts, images, or
impulses that cause anxiety. These thoughts are distressing, and the
individual is unable to ignore them or push them out of their mind.
Compulsions: Repetitive behaviors or mental acts that an individual
feels compelled to perform in response to an obsession. These actions
are performed to reduce anxiety caused by the obsessive thoughts.
OCD Characteristics:
OCD typically begins between the ages of 15 and 25, though it can
develop later in life or even in childhood.
It causes significant distress and impacts daily functioning.
A person may have both obsessions and compulsions, but about 20%
of cases involve only obsessive thoughts without compulsive behaviors.
Common obsessions include concerns about dirt, germs, or fear of
accidents or death.
Compulsions can include behaviors like hand washing, checking locks,
or arranging objects in a specific way to relieve anxiety.
Diagnosis:
OCD is diagnosed based on criteria outlined in the ICD-11.
Essential symptoms include persistent obsessions and/or compulsions,
time consumption (more than 1 hour per day), and significant distress
or functional impairment.
Non-essential symptoms may include temporary relief after performing
compulsions or avoidance of triggering situations.
Characteristics of Obsessions and Compulsions in OCD
Obsessions:
Unwanted thoughts: Intrusive and distressing thoughts or mental
images that repeatedly enter the mind.
Anxiety-inducing: These thoughts cause high levels of anxiety, which
the individual cannot control.
Common themes: Obsessions often involve fears of contamination
(dirt or germs), harm (accidents or death), or the need for perfection
(things being "just right").
Compulsions:
Repetitive behaviors: Actions or mental rituals that the individual
feels compelled to perform to reduce the anxiety caused by the
obsessions.
Relief from anxiety: Performing the compulsion offers temporary
relief from the distress caused by obsessive thoughts.
Strict rules: Compulsions often follow a set of rigid rules, such as
needing to wash hands a specific number of times, or checking locks
repeatedly.
Avoidance: Individuals may avoid people, places, or situations that
trigger obsessive thoughts or compulsive behaviors.
Evaluation
Biological Explanations (Nature):
Strengths:
o Neuroimaging and genetic studies provide evidence that
biological factors, such as damage to the basal ganglia and
specific genetic predispositions (e.g., COMT and SERT genes),
play a role in the development of OCD.
o The heritability estimate of 27-47% supports the idea that
genetics contribute to OCD, highlighting the importance of
biological factors in the disorder.
Weaknesses:
o Not everyone with basal ganglia damage develops OCD,
suggesting that other factors, such as environmental influences,
are important.
o The heritability estimate of around 50% suggests that genes
alone do not account for OCD, and this highlights the need to
consider environmental factors in the development of OCD.
Cognitive Explanations (Nurture):
Strengths:
o Cognitive explanations, such as hypervigilance and thought-
action fusion (TAF), provide insights into the specific thought
patterns that maintain OCD symptoms. Research, including
Rachman’s (2004) case study, supports these cognitive biases
as important factors in OCD.
o CBT, which is based on these cognitive principles, has been
shown to be an effective treatment for OCD by helping
individuals challenge irrational thoughts and reduce compulsive
behaviors.
Weaknesses:
o Much of the research on cognitive explanations is based on case
studies, which limits the ability to generalize findings to the
broader population.
o Cognitive explanations focus primarily on internal thought
processes and do not account for the social and biological
factors that also contribute to OCD. For example, parental
behavior (such as a parent with contamination fears) might
influence a child's development of OCD, but this is not
emphasized in cognitive models.
Overall Conclusion:
Both biological and cognitive explanations provide valuable insights
into OCD. However, they are not mutually exclusive and should be
considered together. While biological factors (e.g., brain damage and
genetics) can predispose individuals to OCD, cognitive processes (e.g.,
faulty thinking patterns and biases) play a significant role in the
development and maintenance of the disorder. Further research is
needed to explore the interaction between biological and cognitive
factors and to develop more comprehensive treatments that address
both aspects.
Systematic Sampling
Definition: Systematic sampling is a method of selecting a sample
from a larger population by choosing every 'nth' individual from the
population.
o For example, in a population of 1,000 people, one might select
every 10th person, resulting in a sample of 100 individuals.
Summary Table for CBT and Evaluation
Evaluation
Importance of the Study:
The study demonstrated that CBT (including ERP) is effective in
treating OCD, even within group settings, significantly reducing
symptoms in individuals with OCD.
One of the key takeaways is that group therapy can make CBT more
accessible, enabling more patients to receive treatment. Group settings
could be a practical alternative, especially when individual therapy
might not be feasible due to time, cost, or availability.
Limitations of the Study:
1. Sample Size: The study had a small sample size, which limits the
generalizability of the results.
2. Selection Bias: The participants were not randomly selected, which
could introduce selection bias. The findings may not apply to a broader,
more diverse population.
3. Lack of Control Group: There was no comparison or control group,
which means it is difficult to determine whether the improvements
were specifically due to the therapy or other factors.
4. No Long-term Follow-up: The study did not measure the long-term
effectiveness of the treatment. Without long-term data, it is unclear
whether the benefits of group therapy are maintained after the therapy
ends.
PTSD
PTSD is a mental health disorder that can develop after an individual has
been exposed to a traumatic event. Unlike temporary negative emotions, the
effects of PTSD can be long-lasting, often interfering with daily life,
relationships, and work. The traumatic event could be either long or short but
must be deeply distressing and frightening in nature.
Three Core Elements of PTSD:
1. Re-experiencing:
This is the re-living of the traumatic event through intrusive memories,
nightmares, and flashbacks. It feels as though the traumatic event is
happening again in the present moment, not just remembering it.
These memories can be mild or so severe that the person loses
awareness of reality. During re-experiencing episodes, the individual
may have intense emotional and physical reactions, feeling exactly
how they did during the event itself.
2. Avoidance:
People with PTSD may intentionally avoid reminders of the traumatic
event. This can include avoiding places, situations, or even people that
are associated with the trauma. It is a way of trying to protect
themselves from the painful process of re-living the traumatic
experience.
3. Hypervigilance:
Hypervigilance refers to a state of constant anxiety, where the person
is on high alert and reacts excessively to everyday stimuli (e.g.,
jumping at a bird’s noise). This extreme anxiety may cause the
individual to adopt behaviors aimed at feeling safer, like checking the
weather repeatedly for signs of disaster or avoiding headphones when
walking alone, as a way to stay alert to their surroundings.
Biological and Cognitive Influences on PTSD:
Biological Factors:
1. Cortisol Imbalance:
o Cortisol is a hormone released by the adrenal glands during
stress as part of the "fight or flight" response. It helps the body
prepare to respond to threats.
o Research has shown that individuals with PTSD can experience
imbalances in cortisol levels:
High cortisol levels: These may be a result of prolonged
activation of the stress response, which can exacerbate
PTSD symptoms.
Low cortisol levels: Low cortisol is also linked to PTSD,
with studies showing that lower levels of this hormone
increase the risk of developing PTSD.
o Evaluation: Testing for cortisol imbalances can help identify
individuals at risk of PTSD, allowing for earlier intervention.
However, it is important to note that cortisol imbalance alone
does not fully explain PTSD, and other factors, including genetics
and life experiences, play a role.
2. Genetics and PTSD:
o A study by Nievergelt et al. (2019) explored the role of genetics
in PTSD by analyzing genetic data from 200,000 individuals
globally. The study found that genetics account for 5-20% of the
risk for PTSD.
o The study suggests that certain gene variants inherited from
parents may predispose individuals to PTSD.
o Evaluation: This research supports the notion of a genetic
vulnerability to PTSD. However, the genetic link may be
influenced by personality traits and environmental factors, as
seen in twin studies where identical twins may not both develop
PTSD despite having the same genes. This highlights the
complex interaction of nature (genetics) and nurture
(environment).
Cognitive Influence: Dual Representation Theory:
Dual Representation Theory (Brewin et al., 1996) explains
PTSD symptoms in terms of two memory systems:
1. Verbally Accessible Memory System (VAM): This system
processes information consciously and can be deliberately
recalled.
2. Situationally Accessible Memory System (SAM): This system
processes information unconsciously and is difficult to recall
intentionally or in an organized manner.
During a traumatic event, the VAM is impaired, while the SAM
continues to process the event. This leads to symptoms such
as flashbacks, where the individual re-lives the trauma
unconsciously.
Emotional Processing Endpoints (Post-Trauma):
1. Completion: Successful recovery where traumatic memories are
processed and integrated.
2. Chronic Emotional Processing: Incomplete integration, leading to
persistent symptoms and potentially secondary conditions (e.g.,
OCD).
3. Premature Inhibition of Processing: Avoidance of trauma
memories, leading to reactivation of symptoms later on.
Evaluation: The Dual Representation Theory is valuable for
understanding PTSD from a cognitive perspective, particularly in the
way memories of trauma are processed. It aligns well with biological
explanations, such as the role of cortisol. However, one criticism is the
unclear interaction between the VAM and SAM systems, and how they
work together during traumatic memory processing. The theory lacks
clarity on how these memory systems communicate.
Prolonged Exposure Treatment for PTSD:
Prolonged Exposure (PE) is a leading therapeutic approach for treating
PTSD, developed by Dr. Edna Foa. The treatment involves confronting
traumatic memories and emotions that individuals with PTSD have
been avoiding.
Four Key Elements of PE:
1. Psychoeducation: Educating clients about the psychological
responses to trauma and how these responses contribute to
PTSD.
2. Breathing Techniques for Relaxation: Teaching clients to control
their breathing to manage emotional distress during therapy.
3. In Vivo Exposure: Gradual exposure to real-life trauma-related
situations (e.g., entering an airport after a plane crash) in a safe
and controlled manner. Over time, clients learn to tolerate the
emotional and physical responses associated with these
triggers.
4. Imaginal Exposure: Clients repeatedly recall and verbalize the
traumatic memories with a therapist, helping to process and
integrate these memories into a non-traumatic framework.
Evaluation:
o Strengths:
Gradual process: The hierarchy-based exposure allows for
personalization, making the treatment flexible and
adaptable for various types of trauma.
It can be combined with other therapies (e.g.,
medication) to enhance effectiveness.
It is now considered one of the most effective treatments
for PTSD.
o Limitations:
If the client is still in a traumatic environment or lacks a
support system, PE can be too risky and may exacerbate
symptoms.
Clients may find recording and listening to sessions
uncomfortable, raising concerns about privacy and
emotional distress.
Willingness: The client must be willing and able to
participate in exposure therapy, which can be challenging
if the trauma is still fresh or overwhelming.
Summary
Eftekhari's study provides strong evidence supporting the effectiveness of
Prolonged Exposure Therapy for chronic PTSD. By reviewing multiple
studies, the research demonstrates that PE can significantly reduce PTSD
symptoms and help individuals regain control over their emotional responses
to trauma. However, the study also highlights gaps in understanding, such as
which aspects of PE are most effective and how to best adapt the treatment
for diverse cultural groups. Despite these limitations, the study offers valuable
insights for both clinicians and researchers seeking to improve PTSD
treatments.