TEST REPORT

Reg. No. : 50104901042Reg. Date : 18-Jan-2025 08:46 Ref.No : Approved On : 18-Jan-2025 11:19
Name : Mr. YASHWANT MALVIYA Collected On : 18-Jan-2025 08:46
Age : 30 Years Gender: Male Pass. No. : Dispatch At :
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Test Name Results Units Bio. Ref. Interval

Complete Blood Count
Specimen: EDTA blood

Hemoglobin
Hemoglobin(SLS method) 14.6 g/dL 13.0 - 17.0
Hematocrit (calculated) 44.8 % 40 - 50
RBC Count(Ele.Impedence) 5.28 X 10^12/L 4.5 - 5.5
MCV (Calculated) 84.9 fL 83 - 101
MCH (Calculated) 27.6 pg 27 - 31
MCHC (Calculated) 32.5 g/dL 32 - 36
RDW (Calculated) 11.5 11.5 - 16.0
Differential WBC count (Impedance and flow)
Total WBC count 8780 /µL 4000 - 10000
Neutrophils H 90 50 - 70
Lymphocytes L 09 20 - 40
Monocytes 01 <10
Eosinophils 00 0-5
Basophils 00 0-2
Platelet
Platelet Count (Manual) 239000 /µL 150000 - 450000
MPV 8.00 fL 6.5 - 12.0
EDTA Whole Blood

Note: All abnormal hemograms are reviewed and confirmed microscopically.Peripheral blood smear and malarial parasite examination are not part of CBC report.

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 TEST REPORT
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Test Name Results Units Bio. Ref. Interval

LIPID PROFILE

CHOLESTEROL 180.00 mg/dL <200 : Desirable,

Enzymetic Colorimetric Method, CHOD-POD
200-239 : Borderline High,
>=240 : High
Triglyceride H 204.10 mg/dL <150 : Normal,
Method:GPO-POD
150-199 : Border Line High,
200-499 : High,
>=500 : Very High
Very Low Density Lipoprotein(VLDL) H 41 mg/dL 0 - 30
Calculated

Low-Density Lipoprotein (LDL) 117.20 mg/dL < 100 : Optimal,

100-129 : Near Optimal/above
optimal,
130-159 : Borderline High,
160-189 : High,
>=190 : Very High
High-Density Lipoprotein(HDL) 21.80 mg/dL <40 Low (High Risk),
Method:Homogeneous Enzymatic Colorimetric
>=60 High(Low Risk)
CHOL/HDL RATIO H 8.26 0.0 - 3.5
Calculated

LDL/HDL RATIO H 5.38 1.0 - 3.4

Calculated

TOTAL LIPID 728.20 mg/dL 400 - 1000

Calculated
Serum

As a routine test to determine if your cholesterol level is normal or falls into a borderline-, intermediate- or high-risk category.
To monitor your cholesterol level if you had abnormal results on a previous test or if you have other risk factors for heart disease.
To monitor your body’s response to treatment, such as cholesterol medications or lifestyle changes.
To help diagnose other medical conditions, such as liver disease.
Note : biological reference intervals are according to the national cholesterol education program ( NCEP) guidelines.

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Test Name Results Units Bio. Ref. Interval

ESR 15 mm/hr 17-50 Yrs : <12,

Method:Modified Westergren
51-60 Yrs : <19,
61-70 Yrs : <20,
>70 Yrs: <30
EDTA Whole Blood

Clinical Biochemistry
FASTING PLASMA GLUCOSE
Specimen: Fluoride plasma
Fasting Plasma Glucose H 135.40 mg/dL Normal: <=99.0
Prediabetes: 100-125
Diabetes :>=126
Flouride Plasma

Criteria for the diagnosis of diabetes:

1. HbA1c >/= 6.5 *
Or
2. Fasting plasma glucose >126 gm/dL. Fasting is defined as no caloric intake at least for 8 hrs.
Or
3. Two hour plasma glucose >/= 200mg/dL during an oral glucose tolerence test by using a glucose load containing equivalent of 75 gm anhydrous glucose dissolved in
water.
Or
4. In a patient with classic symptoms of hyperglycemia or hyperglycemic crisis, a random plasma glucose >/= 200 mg/dL. *In the absence of unequivocal hyperglycemia,
criteria 1-3 should be confirmed by repeat testing. American diabetes association. Standards of medical care in diabetes 2011. Diabetes care 2011;34;S11.

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Test Name Results Units Bio. Ref. Interval

HbA1c 5.8 % Normal: <= 5.6

Method:HPLC
Prediabetes:5.7-6.4
Diabetes: >= 6.5
6-7 : Near Normal Glycemia,
<7 : Goal ,7-8 : Good Control
,>8 : Action Suggested.
Mean Blood Glucose 120 mg/dL
( Calculated )
EDTA Whole Blood

Criteria for the diagnosis of diabetes

1. HbA1c >/= 6.5 * Or Fasting plasma glucose >126 gm/dL. Fasting is defined as no caloric intake at least for 8 hrs. Or
2. Two hour plasma glucose >/= 200mg/dL during an oral glucose tolerence test by using a glucose load containing equivalent of 75 gm anhydrous glucose dissolved in
water. Or
3. In a patient with classic symptoms of hyperglycemia or hyperglycemic crisis, a random plasma glucose >/= 200 mg/dL. *In the absence of unequivocal hyperglycemia,
criteria 1-3 should be confirmed by repeat testing.American diabetes association. Standards of medical care in diabetes 2011. Diabetes care 2011:34:S11.
Limitation of HbA1c
1) In patients with Hb variants even analytically correct results do not reflect the same level of glycemic control that would be expected in patients with normal
population.
2) Any cause of shortened erythrocyte survival or decreased mean erythrocyte survival or decreased mean erythrocyte age eg. hemolytic diseases, pregnancy, significant
recent/chronic blood loss etc. will reduce exposure of RBC to glucose with consequent decrease in HbA1c values.
3) Glycated HbF is not detected by this assay and hence specimens containing high HbF (>10%)may result in lower HbA1c values than expected. Importance of HbA1C
(Glycated Hb.) in Diabetes Mellitus
- HbA1C, also known as glycated heamoglobin, is the most important test for the assessment of long term blood glucose control( also called glycemic control).
- HbA1C reflects mean glucose concentration over pas 6-8 weeks and provides a much better indication of longterm glycemic control than blood glucose determination.
- HbA1c is formed by non-enzymatic reaction between glucose and Hb. This reaction is irreversible and therefore remains unaffected by short term fluctuations in blood
glucose levels.
- Long term complications of diabetes such as retinopathy (Eye-complications), nephropathy (kidney-complications) and neuropathy (nerve complications), are potentially
serious and can lead to blindness, kidney failure, etc.
- Glyemic control monitored by HbA1c measurement using HPLC method (GOLD STANDARD ) is considered most important. (Ref. National Glycohaemoglobin
Standardization Program - NGSP)
Note : Biological reference intervals are according to American Diabetes Association (ADA) Guidelines.

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Test Name Results Units Bio. Ref. Interval

KIDNEY FUNCTION TEST

Urea 28.6 mg/dL <= 65 YEARS AGE: <50

UREASE/GLDH
mg/dL;
>65 YEARS AGE: <71 mg/dL
Uric Acid (UA) 6.80 mg/dL 3.4 - 7.0
Uricase

Creatinine H 1.37 mg/dL 0.70 - 1.20

MODIFIED JAFFE KINETIC
Serum

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Test Name Results Units Bio. Ref. Interval

LIVER FUNCTION TEST

LIVER FUNCTION TEST

TOTAL PROTEIN 7.46 g/dL 6.4 - 8.3
Biuret Colorimetric

ALBUMIN 3.70 g/dL 3.2 - 5.0

Bromcresol Green(BCG)

GLOBULIN H 3.76 g/dL 2.4 - 3.5

Calculated

ALB/GLB L 0.98 1.2 - 2.2

Calculated

SGOT 28.40 U/L 0 - 32

Uv Without Pyridoxal 5-Phosphate Activation,IFCC

SGPT H 38.70 U/L 0 - 33

Uv Without Pyridoxal 5-Phosphate Activation,IFCC

Alkaline Phosphatase 75.00 U/L 40 - 130

ENZYMATIC COLORIMETRIC IFCC, PNP, AMP BUFFER

TOTAL BILIRUBIN 0.47 mg/dL 0.0 - 1.2

Diazo

DIRECT BILIRUBIN 0.20 mg/dL 0 - 0.3

Diazo Reaction

INDIRECT BILIRUBIN 0.27 mg/dL 0.0 - 1.00

Calculated

GGT 58.80 U/L 10 - 71

L-Y-Glutamyl-3 Carboxy-4-Nitroanilide, Enzymetic Colorimetric
Serum

CLINICAL SIGNFICANCE:
- This test panel assesses the functional activity of the liver. It is used for screening for liver damage is taking a drug that may
affect the liver.
- Liver function tests check the levels of certain enzymes and proteins in your blood. Levels that are higher or lower than normal
can indicate liver problems.
- Monitor the progression of a disease, such as viral or alcoholic hepatitis, and determine how well a treatment is working
- Measure the severity of a disease, particularly scarring of the liver (cirrhosis), Monitor possible side effects of medications.

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Test Name Results Units Bio. Ref. Interval

THYROID FUNCTION TEST

T3 (triiodothyronine), Total 0.85 ng/mL 0.6 - 1.81

CHEMILUMINESCENCE

T4 (Thyroxine),Total 12.01 µg/dL 4.5 - 12.6

CHEMILUMINESCENCE

TSH (Thyroid stimulating hormone) 1.679 µIU/mL 0.55 - 4.78

CHEMILUMINESCENCE
Serum

Comments:
Thyroid stimulating hormone (TSH) is synthesized and secreted by the anterior pituitary in response to a negative feedback mechanism involving
concentrations of FT3 (free T3) and FT4 (free T4). Additionally, the hypothalamic tripeptide, thyrotropin-relasing hormone (TRH), directly stimulates TSH
production. TSH stimulates thyroid cell production and hypertrophy, also stimulate the thyroid gland to synthesize and secrete T3 and T4. Quantification of
TSH is significant to differentiate primary (thyroid) from secondary (pituitary) and tertiary (hypothalamus) hypothyroidism. In primary hypothyroidism, TSH
levels are significantly elevated, while in secondary and tertiary hypothyroidism, TSH levels are low.
TSH levels During Pregnancy :
• First Trimester : 0.1 to 2.5 µIU/mL
• Second Trimester : 0.2 to 3.0 µIU/mL
• Third trimester : 0.3 to 3.0 µIU/mL
Referance : Carl A.Burtis,Edward R.Ashwood,David E.Bruns. Tietz Textbook of Clinical Chemistry and Molecular Diagnostics. 5th Eddition. Philadelphia: WB
Sounders,2012:2170

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Test Name Results Units Bio. Ref. Interval

MAGNESIUM

Magnesium (Mg) 2.14 1.60 - 2.60

Serum

Magnesium, along with potassium, is a major intracellular cation. Magnesium is a cofactor of many enzyme systems. All adenosine triphosphate (ATP)-dependent
enzymatic reactions require magnesium as a cofactor. Approximately 70% of magnesium ions are stored in bone. The remainder is involved in intermediary metabolic
processes; about 70% is present in free form while the other 30% is bound to proteins (especially albumin), citrates, phosphate, and other complex formers. The serum
magnesium level is kept constant within very narrow limits. Regulation takes place mainly via the kidneys, primarily via the ascending loop of Henle. Conditions that
interfere with glomerular filtration result in retention of magnesium and, hence, elevation of serum concentrations. Hypermagnesemia is found in acute and chronic renal
failure, magnesium overload, and magnesium release from the intracellular space. Mild-to-moderate hypermagnesemia may prolong atrioventricular conduction time.
Magnesium toxicity may result in central nervous system (CNS) depression, cardiac arrest, and respiratory arrest. Conditions that have been associated with
hypomagnesemia include chronic alcoholism, childhood malnutrition, lactation, malabsorption, acute pancreatitis, hypothyroidism, chronic glomerulonephritis,
aldosteronism, and prolonged intravenous feeding.

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Test Name Results Units Bio. Ref. Interval

Calcium 8.20 mg/dL 8.2 - 10.2

Method:BAPTAS

Useful for- Diagnosis and monitoring of a wide range of disorders including diseases of bone, kidney, parathyroid gland or gastrointestinal tract.
Decreased calcium (hypocalcemia)
- Absence or impaired function of parathyroid gland.
- Impaired vitamin d synthesis
- Chronic renal failure
- Hypoalbuminemia
Increased calcium (hypercalcemia)
- Primary hyperparathyroidism
- Bone metastatsis of carcinomas

Inorganic phosphorous (po4) 4.35 mg/dL 2.7 - 4.5

Phosphomolybdate Formation

Serum
Useful for diagnosis and management of a variety of disorders including bone, parathyroid, and kidney disease. Of the phosphorus contained in the body, 88% is
localized in bone in the form of hydroxyapatite. The remainder is utilized during intermediary carbohydrate metabolism and bound to physiologically important
substances such as phospholipids, nucleic acids, and adenosine triphosphate (ATP). Phosphorus exists in blood in the form of inorganic phosphate and organically
bound phosphoric acid. Serum phosphate concentrations are dependent on dietary intake and regulation by hormones such as parathyroid hormone (PTH) and
1,25 vitamin D, and systemic acid base status. Hypophosphatemia may have 4 general causes: shift of phosphate from extracellular to intracellular, renal
phosphate wasting, loss from the gastrointestinal tract, and loss from intracellular stores. Hyperphosphatemia is usually secondary to an inability of the kidneys to
excrete phosphate and is common in patients with chronic kidney disease. Acute hyperphosphatemia can occur as a result of tissue breakdown such as
rhabdomyolysis. Phosphorus has a very strong biphasic circadian rhythm. Values are lowest in the morning, peak first in the late afternoon and peak again in the
late evening. The second peak is quite elevated and results may be outside the reference range.

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Test Name Results Units Bio. Ref. Interval

ELECTROLYTES

Sodium (Na+) 140.10 mmol/L 136 - 145

Method:ISE

Note:
- Sodium is the primary extracellular cation. Sodium is responsible for almost one half the osmolality of the plasma and therefore plays a central role in maintaining
the normal distribution of water and the osmotic pressure in the extracellular fluid compartment.
- Sodium assays are important in assessing acid-base balance, water balance, water intoxication, and dehydration.

Potassium (K+) 4.2 mmol/L 3.5 - 5.1

Method:ISE

Note:
- Potassium is the major cation of the intracellular fluid.
- Used in evaluation of electrolyte balance, cardiac arrhythmia, muscular weakness, hepatic encephalopathy, and renal failure
- Potassium should be monitored during treatment of many conditions but especially in diabetic ketoacidosis and any intravenous therapy for fluid replacement.

Chloride(Cl-) 106.72 mmol/L 98 - 107

Method:ISE

Note:
Chloride is the major anion in the extracellular water space. In normal individuals, serum chloride values vary little during the day, although there is a slight
decrease after meals due to the diversion of chloride to the production of gastric juice.

Comments
The electrolyte panel is ordered to identify electrolyte, fluid, or pH imbalance. Electrolyte concentrations are evaluated to assist in investigating conditions that cause
electrolyte imbalances such as dehydration, kidney disease, lung diseases, or heart conditions. Repeat testing of the electrolyte or its components may be used to
monitor the patient's response to treatment of any condition that may be causing the electrolyte, fluid or pH imbalance.

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Immunoassay
Prostate Specific Antigen (PSA),Total 0.740 ng/mL 0-4
CHEMILUMINESCENCE

Serum
Useful For
1. Evaluating patients with documented prostate problems in whom multiple prostate-specific antigen tests may be necessary per year
2. Monitoring patients with a history of prostate cancer as an early indicator of recurrence and response to reatment.
3.Prostate cancer screening.
Comments
-Prostate-specific antigen (PSA) is a glycoprotein that is produced by the prostate gland, the lining of the urethra, and the bulbourethral gland. Normally, very little
PSA is secreted in the blood. Increases in glandular size and tissue damage caused by benign prostatic hypertrophy, prostatitis, or prostate cancer may increase
circulating PSA levels.
-Digital rectal examination generally does not increase normal prostate-specific antigen (PSA) values. However, cystoscopy, urethral instrumentation, and prostate
biopsy may increase PSA levels.

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Test Name Results Units Bio. Ref. Interval

25 OH Vitamin D L 16.71 ng/mL Deficiency : <10

Insufficiency : 10 - 30
Sufficiency : 30 - 100
Toxicity : >100
Method: CLIA

Vitamin D is a fat soluble hormone involved in the intestinal absorption and deregulation of calcium. It is synthesized by skin when sunlight strikes bare skin. It can
also be ingested from animal sources. Vitamin D is bound to the binding protein (albumin and vitamin D binding protein) and carried to the liver. In the liver it is
transformed in to 25 hydroxy-vitamin D (calcidiol), which is the primary circulating and the most commonly measured form in serum. Then in the kidney it is
transformed in to 1,25 dihydroxy-vitamin D (calcitriol), which is the biologically active form.
Vitamin D plays a vital role in the formation and maintenance of strong and healthy bones. Vitamin D deficiency has long been associated with rickets in children
and osteomalacia in adults. Long term insufficiency of calcium and vitamin D leads to osteoporosis. There have been multiple publications linking vitamin D
deficiency to several disease states, such as cancer, cardiovascular disease, diabetes, and autoimmune diseases.

Vitamin B12 L 110.00 120 - 911

Serum
INTERPRETATION
Dietary sources of Vitamin B12 are meat, eggs, milk and milk products. Vitamin B12 requires intrinsic factor for absorption from intestine.
B12 deficiency causes hematological and neurological abnormalities. Decreased serum B12 levels causes increased excretion of methylmalonic acid. The impaired
DNA synthesis associated with Vitamin B12 deficiency causes macrocytic anemias. In sever is characterized by abnormal maturation of erythrocyte, myeloid
precursors and megakaryocytes in the bone marrow, which results in the pancytopenia.
Withhold Vitamin B12 injecion before the blood is drawn. Blood collected after Vitamin B12 Injection interfere with result. Preservatives such as fluorides &
ascorbic acid interfere with this assay. Excessive exposure of the specimen to light may alter Vitamin B12 result.

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Test Name Results Units Bio. Ref. Interval

IRON PROFILE WITH TRANSFERRIN SATURATION

Iron (Fe) 49.3 µg/dL 49 - 181

Ferrozine

TIBC 289.3 µg/dL 250 - 450

Calculated

Transferrin Saturation L 17.04 % 20 - 60

Calculated
Serum

Note:
Iron determinations are performed for the diagnosis & monitoring of treatment of all the microcytic hypochromic diseases such as Iron deficiency anemia,
hemochromatosis & chronic renal disease as well as macrocytic anemia & also normocytic anemia, hemolytic anemia, hemoglobinopathies, bone marrow
disease & toxic bone marrow damage. Interpretation of iron status must be correlated with other parameters given below as a whole studies rather than
interpreting a single test.
1. IRON DEFICIENCY ANEMIA: S. Iron (low), TIBC (high), Transferrin saturation (low), Ferritin (low), HB Electrophoresis Normal. Smear (Micro/hypo)
2. ANEMIA OF CHRONIC DISEASE: S. Iron (low), TIBC (low), Transferrin saturation (low), Ferritin (Normal/High), HB Electrophoresis (Normal). Smear
(Micro/hypo/Normal)
3. THALASSEMIA: S. Iron (Normal/High), TIBC (Normal), Transferrin Saturation (Normal/High), Ferritin (High), HB Electrophoresis Abnormal.Smear
(Micro/hypo with target cells)
4. SIDEROBLASTIC ANEMIA: S. Iron (Normal/High), TIBC (Normal/High), Transferrin Saturation (Normal/High), Ferritin (High), HB Electrophoresis Normal.
Smear micro/hypo/variable)
5. IRON OVERLOAD(HEMOCHROMATOSIS): S. Iron(High), TIBC(low), Transferrin saturation(High), Ferritin(High), HB Electrophoresis Normal, smear
(variable)
6. MEGALOBLASTIC ANEMIA:.S. Iron(High), TIBC(low), Transferrin saturation(High), Ferritin(High), HB Electrophoresis Normal, smear(Macrocytic)
(Harrison's principles of internal medicine vol.-1, 15th edition/663)

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Test Name Results Units Bio. Ref. Interval

URINE ROUTINE EXAMINATION

Physical Examination
Colour Pale Yellow
Clarity Slight Turbid
CHEMICAL EXAMINATION (by strip test)
pH 6.00 4.6 - 8.0
Sp. Gravity 1.010 1.003 - 1.030
Protein Negative Nil
Glucose Negative Nil
Ketone Negative Nil
Bilirubin Nil Nil
Nitrite Negative
Leucocytes Nil 0-5
Blood Present(++) Absent
MICROSCOPIC EXAMINATION
Leucocytes (Pus Cells) 6 -8/hpf 0 - 5/hpf
Erythrocytes (RBC) 8 - 10/hpf 0 - 5/hpf
Casts Nil /hpf Absent
Crystals Nil Absent
Epithelial Cells 3 - 5/hpf Nil
Monilia Nil Nil
T. Vaginalis Nil Nil
Bacteria Present Absent
Urine

Remarks: Urinalysis : microscopic examination is carried out on centrifuged urinary sediment.

Urine protein rechecked and confirmed by sulphosalicyic acid.

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Test done from collected sample.

Page 14 of 14
Approved by: Dr. Dipika Jaiswal
Generated On : 18-Jan-2025 14:38 M.D. Pathology
Approved On: 18-Jan-2025 14:12