Tanner Staging
Tanner Staging
It was developed by Marshall and Tanner while conducting a longitudinal study during the 1940s to the
1960s in England.
Based on observational data, they developed separate scales for:
I. the development of external genitalia: phallus, scrotum, and testes volume in males;
II. Breasts in females
III. Pubic hair in both males and females.
Function
- monitor for deviations in normal timing and sequence of physical signs of puberty that may represent physiologic
problems.
- idiopathic conditions
- nutritional deficiencies,
II
Development of a breast bud, with elevation of the
papilla and enlargement of the areolar diameter.
III
Enlargement of the breast, without separation of
areolar contour from the breast
IV
The areola and papilla project above the breast,
forming a secondary mound.
V
Recession of the areola to match the contour of
the breast; the papilla projects beyond the contour
of the areola and breast.
Breastfeeding Policy
CWMH Policy
® If medically indicated
® If mother has a fully formed choice after fully formed choice
receiving counseling on various options and risk & benefits of
each.
® Reasons for supplemental and replacement feeds are
documented
7. Step 7
A00071202
Sarah Sharon Narayan
BREAST ENGORGEMENT
• Engorgement — occurs either from interstitial edema with the onset of lactation after birth, or at other times
during lactation with accumulation of excess milk.
Etiology
• Primary: the engorgement occurs with the onset of copious milk production (ie, lactogenesis stage II), usually
between days three to five after delivery. It is due to interstitial edema of the breast prompted by the decrease in
progesterone levels after the placenta is delivered. (Transition from colostrum to mature milk)
• Secondary: typically occurs later when there is a mismatch between milk production and extraction as the
mother's milk supply exceeds the amount of milk removed by her infant. This may occur from excessive
stimulation of milk production via pumping, taking medications that increase milk supply, or decreased milk
extraction resulting in insufficient removal of breast milk (e.g., due to infrequent feeds, poor attachment,
ineffective suckling, abrupt cessation of breastfeeding).
• Breast firmness and fullness • Some mothers may find use of a breast pump or hand
expression to be helpful. However, this use should be
• Pain and tenderness
limited to immediately before a feeding to soften the breast
How do we manage? because overuse will stimulate milk production and could
• Effective management hinges on adequate removal increase the engorgement.
of the milk. For primary engorgement, it is • For others, applying pressure toward the chest wall softens
important to ensure implementation of good feeding the edema and facilitates the latch.
techniques with a satisfactory latch and optimal
nursing positioning.
• If the areola is involved, manual expression of
small amounts of milk before the feeding will
soften the areola and facilitate latching
• Applying warm compresses or a warm shower enhances let-down and may facilitate milk removal either by hand
expression or with suckling
• After or between feedings, cold compresses may decrease the swelling and discomfort
• Analgesics such as ibuprofen and acetaminophen may decrease the discomfort
• Topical application of cool green cabbage leaves is soothing, inexpensive, and unlikely to be harmful
• Engorgement usually resolves over time.
• Complication of engorgement: mastitis
LACTATIONAL MASTITIS (MILK
FEVER)
• Lactational mastitis-breast becomes painful, swollen, and red ( inflammation of the breast parenchyma)
• it is most common in the first three months of breastfeeding. (4-6 weeks postpartum) in approximately 10% of
nursing mothers.
• tends to occur in the setting of breastfeeding problems that result in prolonged engorgement or poor drainage,
including partial blockage of milk duct, inefficient milk removal or infrequent feedings, oversupply of milk,
nipple trauma, and pressure on the breast
• Initially, engorgement occurs because of poor milk drainage, probably related to nipple trauma with
resultant swelling and compression of one or more milk ducts.
• If symptoms persist beyond 12 to 24 hours, the condition of infective lactational mastitis develops (since breast
milk contains bacteria); this is characterized by pain, redness, fever, and malaise
ETIOLOGY
• Partial blockage of milk duct; reduced drainage results in stagnant milk distal to the
obstruction
• Oversupply of milk
• Infrequent feedings
• Nipple excoriation or cracking
• Rapid weaning
• Illness in mother or baby
• Maternal stress or excessive fatigue
• Maternal malnutrition
PATHOPHYSIOLOGY
• Nipple fissures facilitate the entry of bacteria located in the nostril and throat of the infant or on the skin of the mother
into the milk ducts during breastfeeding.
• Prolonged breast engorgement (due to overproduction of milk ) or insufficient drainage of milk (e.g., due to infrequent
feeding, quick weaning, illness in either the baby or mother) result in milk stasis, which creates favorable conditions
for bacterial growth within the lactiferous ducts.
It is a clinical diagnosis. Breast milk cultures can be done if mother is unresponsive to treatment, infection is severe or if it
keeps recurring. If mom shows signs of sepsis/ systemic infection, blood cultures can be performed.
TREATMENT
• In nursing mothers, breastfeeding with alternate breasts is recommended every 2–3 hours. ( no need to stop breastfeeding)
• Analgesics (e.g., ibuprofen)
• Cold compresses
• Antibiotic treatment- Oral penicillinase-resistant penicillin or cephalosporin (e.g., dicloxacillin or cephalexin)
• In the case of methicillin-resistant Staphylococcus aureus (MRSA): clindamycin (risk of Clostridioides difficile colitis) or
trimethoprim-sulfamethoxazole (septrin not for <1 month old) or vancomycin (systemic illness)
• Tx for 5-7 days or 10-14 days
• In the case of inadequate response to initial treatment:
Initiate treatment according to breast milk culture results.
• Consider an underlying breast abscess, which requires surgical drainage.
Prevention
• Anticipatory lactational counseling
• To prevent recurrence: oral Lactobacillus probiotic
BREAST
PROBLEMS
BREAST ABSCESS
CANDIDA MASTITIS
BREAST ABSCESS
– It is a painful build-up of pus in the breast caused by infection.
– Breast infections are divided into: Lactational and Non-lactational or Puerperal and Non-
puerperal.
– Breast abscesses are more common in lactating women.
– Clinical manifestation:
History of breast pain, erythema, warmth, and possibly edema.
Complain of fever, nausea, and vomiting, purulent drainage from the nipple, or the site
of erythema.
– Etiology of breast abscess:
Lactational breast abscess: Staphylococcus aureus and Streptococcal species including
methicillin-resistant S. aureus.
Non-lactational breast abscess: Result of mixed flora with S. aureus, Streptococcus, and
anaerobic bacteria.
CANDIDA MASTITIS – BREAST AND NIPPLE THRUST
– Thrush is a fungal infection caused by the organism Candida albicans, which can occur in the
nipples or breast tissue.
– Nipple pain that doesn’t go away even upon adjustment of breastfeeding attachment, may
indicate a breast problem.
– Breast and nipple thrush may be linked to a history of vaginal thrush, recent use of antibiotics or
nipple damage.
BREAST PROBLEMS
NIPPLE PAIN
Etiology:
Nipple injury:
Fissured nipple
●Normal nipple sensitivity
●Nipple injury- poor position or latch on, harsh breast
cleaning, infant biting, use of irritating products
● Areolar dermatitis
●Breast engorgement
Areolar dermatitis:
●Plugged ducts During an acute presentation,
vesicles, crusting, and erosions
●Nipple and breast infections are seen in the affected
areas (panel A),
●Excessive milk supply whereas in the chronic state, the
areas are generally dry,
erythematous and scaling (panel
B).
CARE OF TRAUMATIZED NIPPLES