Module 4
Module 4
DIGESTIVE SYSTEM-IV
LEARNING OBJECTIVES
Definition
Enteritis is inflammation of the whole of the intestinal tract. But
usually it is applied to the inflammation of the small intestines.
The inflammation of the colon is called colitis , that of cecum
typhlitis, that of rectum proctitis and of cloca cloacitis or vent gleet.
Introduction
Enteritis is of immense economic importance.
Occurrence
Enteritis is very common in domesticated animals and fowls.
Since enteritis occurs along with gastritis (the same irritants
causing gastritis passing on to intestines produces enteritis also)
gastro-enteritis is a frequent condition met with.
Etiology
Causes are many and varied and they include chemicals, bacteria,
viruses, protozoa, rickettsia, helminths, fungi, , disturbed metabolic
processes as in ruminants, venous congestion as in portal
hypertension and congestive cardiac failure, toxins of Clostridia,
coliforms and spoiled or mouldy feeds and avitaminosis.
Gross pathology
In enteritis, the whole length
of the bowel may not be affected,
inflammation localizing only at
one part or the other.
TYPES OF ENTERITIS
Predisposing causes
In calves and lambs avitaminosis A is a predisposing factor while in
young pigs deficiency of animal proteins and trace elements
predispose them to infections.
In such a state, the organisms are able to gain a foothold and thrive
causing the disease.
Aetiology
Causes include mild irritants like foreign bodies, sand, coarse feeds,
chemicals, bites of parasites (hook worms), Vibrio coli (causing winter
diarrhoea in cattle) and drugs.
Acute catarrhal enteritis may be noticed in
• Enteritis in sucklings – scours in calves, lambs, foals and piglets caused by
E.coli, Pasteurella, Salmonella, Proteus, Vibrio and Streptococci.
• Enterotoxemia in sheep.
• Viral Diarrhoea -Mucosal Disease in cattle.
• Virus gastroenteritis in pigs.
• Salmon poisoning in dogs.
• Salmonellosis, infectious cloacitis, pullet disease and ornithosis in fowls.
• Oral antibiotic therapy may cause enteritis in two ways
These may themselves be irritants or
They may so alter the intestinal flora that there is over growth of other
bacteria (Staphyloccocci, Proteus sp; Pseudomanas sp) and fungi (Candida
albicans) which are normally kept under restraint and so enteritis results.
Immature amphistomes
One should be able to distinguish this condition from the normal hyperemia
that occurs during active digestion.
The intestinal contents are watery
the mucosa is reddish in color and slightly thickened, covered with a
mucinous exudate.
The peyer’s patches are prominent being hyperplastic, outlined by a zone of
hyperemia.
HISTOPATHOLOGY
Aetiology
It may develop from the acute condition or more usually it may
arise gradually as in Johne’s disease, intestinal helminthiasis, chronic
venous congestion (due to congestive cardiac failure) and cirrhosis of
liver.
Gross pathology
The wall of the intestines is
greatly thickened.
The mucosa is smooth
(covered by thick mucus)
The corrugations are
sometimes present at right
angles to the length of the
intestines (Chronic polypoid
enteritis).
HISTOPHALOGY