• Normal labouris characterized by
coordinated uterine contractions
associated with progressive dilation
of cervix and descent of fetal head.
• Associated with cervical dilatation
≥ 1 cm /hr in Nulliparous woman
• Likely to end in successful vaginal
delievery.
3.
Normal Uterine Contractions:-
❖Polarityof Uterus
➢ When the upper segment contracts,
the lower segment relaxes
❖Pacemakers
➢ There are two pacemakers
➢ situated at each cornua of the uterus
➢ Generates uterine contractions
in a coordinated fashion
4.
Properties of NormalUterine Contractions:-
➢ The intensity of contraction
diminishes from top to bottom of the
uterus
➢ The contraction waves starts of the
pacemaker and propogates towards the
lower uterine segment
➢ The duration of contraction
diminishes progressively as the wave
moves away from the pacemaker
❖In dysfunctional labor, new
pacemaker may come up anywhere in
5.
DEFINITION:-
of the normal
“Anydeviation
pattern contractions
affecting
of uterine
the course
of
designated as disordered
labour is
or
abnormal uterine action.”
6.
➢Effective Uterine Contractionsstrats
at the cornua and gradually sweeps
downwards over the uterus.
➢In Primary Dysfunctional Labor,
Uterine Activity instead of being
governed by a single dominant
pacemaker, is shifted to less
efficient contractions due to
emergence of other pacemaker foci.
➢ Oxytocin therapy may be effective in
restoring the global and effective uterine
contractions.
7.
<
➢Primary Dysfunctional Labor,is
defined when the cervix dilates
1cm/hr following a normal latent
phase of labor.
➢ Commonest abnormality
➢ Mostly corrected by,
✓Amniotomy or/and
✓Oxytocin Augmentation
8.
➢ Secondary Arrest,is defined when
the cervical dilatation stops or slows
after the active phase of labour has
started normally.
9.
❑ Uterine activityis measures
by noting...
▪ Basal tone
▪ Active (peak) pressure
▪ Frequency
10.
❑Assesment is usuallydone by...
▪ Clinical Palpation (inaccurate)
▪ Tocodynamometer with
external transducer
▪ Using intrauterine pressure
catheter (accurate)
❖Normal baseline tonus is between
5 and 20 mm of Hg and peak pressure
is around 60 mm of Hg
• Common butcomparatively less serious
• May complecate at any stage of labour
• May be present from beginning of labour
or develop subsequently after a variable
period of effective contraction
19.
Uterine Contractions...
❑The intensityis diminished
❑ Duration is shortened
❑ Good relaxation inbetween contractions
❑ Intervals are increased
❖General pattern of uterine contractions
of labor is maintained but intrauterine
pressure during contraction is below 25 mm
of Hg.
20.
Diagnosis...
❑ Patient feelsless pain during contraction
❑Hand placed over the uterus during
uterine contraction reveals less hardening of
the uterus
❑Uterine wall is easily indentable at
the acme of a pain
❑ Uterus remains relaxed after
contraction
❑ Fetal parts are well palpable
❑ Fetal heart rate remains normal
21.
Internal Examination reveals...
❑Poor dilation of the cervix
❑Associated presence of contracted
pelvis, malposition, deflexed head or
malpresentation
❑ Membranes usually remains intact
Effects on mother and fetus...
❑ Maternal Exhaustion
❑Fetal Distress , are unusual and
appear late.
22.
Management...
❑Case is reassessedto exclude CPD
or Malpresentation
Place of Cesarean Section...
❑ Presence of Contracted Pelvis
❑ Malpresentation
❑ Evidences of fetal or maternal
distress
23.
Vaginal Delivery...
❖Genral Measures:-
➢Keep up the morale of patient
➢ Manage maternal stress and emotion
➢ Avoid supine position
➢ Empty the bladder ( catheterization)
➢Maintain hydration by infusion
of Ringer's solution
➢ Adequate pain relief
24.
Vaginal Delivery...
❖ ActiveMeasures:-
➢Acceleration of uterine contraction
by low rupture of the membrane followed
by oxytocin drip
➢ The drip rate is gradually increased
until effective contractions are set up
➢ The drip is to be continued till one
hour after delivery
• Usually appearsin active stage of
labour.
• The hypertonic state of the uterus
arises from any of the conditions such
as spastic lower uterine segment,
colicky uterus, asymmentrical uterine
contraction, contriction ring or
generalized tonic contraction of the
uterus and all thes states are
collectively called incoordinate uterine
contraction.
27.
• Increased frequencyand or duration of
uterine contractions cause rise in
baseline tone and thereby diminish
circulation in the placental intervillous
space.
• New pacemakers appear all over the
uterus.
• The myometrium contracts spasmodically
and irregularly.
28.
• These contractionsforce neither dilates
the cervix nor pushes the fetus down.
• Uterine tonus is elevated.
• Pain is present before, during and after
contraction.
• This results in fetal hypoxia in labour.
• Placental abruption is often associated
with high baseline tone ( >25 mm Hg ).
29.
• On cardiotocography(CTG) the FHR
shows reduced variability and late
decelerations.
• Uterine hyperstimulation due to oxytocics
are often associated with fetal tachycardia
due to fetal stress.
• Constriction ring, generalized tonic
uterine contraction and cervical dystocia
have got their own separate clinical entity
and as such will be discussed separately.
30.
SPASTIC LOWER SEGMENT
UterineContractions...
❑Fundal dominance is lacking and
often there is reversed polarity
❑ The pacemakers do not work in
rhythm
❑The lower segment contractions
are stronger
❑Inadequate relaxation in
between contractions
❑Basal tone is raised above the critical
level of 20 mm Hg
31.
Diagnosis...
❑The patient isin agony with
unbearable pain reffered to the back
❑There are evidences of dehydration
and ketoacidosis
❑Bladder is frequently distended and
often there is retention of urine, distension
of stomach and bowels are visible
❑There are premature attemps of bear
down
32.
❑ Abdominal palpationreveals:
a) Uterus is tender and gentle
manipulation excites hardening of the
uterus with pain
b)Palpation of the fetal parts is difficult
❑ Internal examination may reveal :
a)Cervix with thick, edematous hangs
loosely like a curtain, not well appliedto the
presenting part
b) Inappropriate dilation of the cervix
c) Absence of mebranes
d)Meconium stained liquor amnii may
ne there
33.
Effect on thefetus...
❑Fetal distress appears early due to
placental insuffiency caused by inadequate
relaxation of the uterus
34.
Management...
❑There is noplace of oxytocin
augmentation with this abnormality
❑Cesarean section is done in majority
of cases
❑Prior correction of dehydration and
ketoacidosis must be achieved by rapid
infusion of Ringer's solution
➢ It isone one form of incoordinate uterine
action where there is localized
myomatrial contraction forming a ring of
circular muscle fibres of the uterus
➢ It is usually situated at the junction of
the upper and lower segment around a
constricted part of the fetus usually
around the neck in cephalic
presentation
➢ It may appear in all the stages of
labour
➢ It is usually reversible and complete
Diagnosis...
❑Difficult
❑Revealed during cesareansection in the
first stage of labour, during forcep application
in second stageand during manual removal in
the third stage
❑The ring is not felt per abdomen
❑Maternal condition is not much affected
but the fetus is in jeopardy because of the
hypertonic state
❑Uterus never ruptures
39.
Treatment...
❑Delivery is usuallydone by
cesarean section
❑The ring usually passes off ny
deepening the plane of anesthesia,
otherwise the ring may to be cut vertically
to deliver the baby
❑The difficlties faced during forceps
delivery or during normal removal of
placenta can be overcome by using deep
anesthesia that relaxes the constriction ring
➢ Progressive cervicaldilatation needs an
effective stretching force by the
preseting force by presenting part
➢ Failure of cervical dilatation may be due
to :
a) Insufficient uterine contractions
b)Malpresentation, Malposition
(abnormal relationship between the
cervix and the presenting part)
➢ Cervical dytocia may be primary or
secondary
42.
Primary cervical dystocia...
❑Commonlyobserved during the...
i. First birth where the external os fails to
dilate
ii. Rigid cervix
iii. Insufficient uterine contractions
iv. others
43.
Treatment...
❑In presence ofassociated
complications (malpresentation,
malposition) cesarean section is preferred
❑If the head is sufficiently low down with
only thin rim of cervix left behind, the rim may
be pushed up manually during contraction or
retraction is given by ventouse
❑In others where the cervix is very much
thinned out but only half dilated, Duhrssen's
incision at 2 and 10 O'clock positions
followed by forceps or ventouse extraction is
quite safe and effective
44.
Secondary cervical dystocia...
❑Thistype of cervical dystocia results
usually due to excess scarring or rigidity
of the cervix from the effect of previous
operation or disease
❑Others are:
i. Post delivery
ii. Postoperative scarring
iii. Cervical cancer
➢ In thiscondition, pronounced retraction
occurs involving whole of the uterus up to
the level of internal os
➢ Thus, there is no physiological
differentiation of the active upper
segment and the passive lower segment
of the uterus
➢ The whole uterus undergoes a sort of
tonic muscular spasm holding the fetus
inside (active retention of the fetus)
➢ Usually there is no risk of rupture
uterus
Clinical features...
❑The patientis in prolonged labour,
having severe and continuous pain
❑Abdominal examination reveals the
uterus to be somewhat smaller in size,
tense and tender
❑Fetal parts are neither well defined, nor
is the fetal heart sound audible
❑Vaginal examination reveals jammed
head with big caput, dry and adematous
vagina
49.
Treatment...
❑Correction of dehydrationand
ketoacidosis by rapid infusion of Ringer's
solution
❑Antibiotic
❑Adequate pain relief
❑Hypercontractility (tachysystole) induced
by oxytocics can be managed by tocolytics.
Oxytocin infusion should be stopped
❑esarean delivery is done in majority of
the cases specially when obstruction is
suspected
“ A labouris called precipitate when the
combined duration of the first and second
stage is less than two hours”
➢ It is common in multiparae and
be repetitive
➢ Rapid expulsion is due to the
combined effect of hyperactive uterine
contractions associated with diminished
soft tissue resistance
➢ Labour is short as rate of
cervical dilatatiion is 5 cm/hour or
more in nulliparous women
52.
Maternal risk...
1. Extensivelaceration of the cervix, vagina
and perineum
2. PPH due to uterine hypotonia that
develops subsequent to unusual vigorous
contractions
3. Inversion
4. Uterine rupture
5. Infection
6. Amniotic fluid ambolism
53.
Fetal risk...
1. Intracranialstress and hemorrhage
because of rapid expulsion without time
for moulding of the head
2. The baby may sustain serious injuries if
delivery occurs in standing position,
bleeding from the torn cord and direct hit
on the skull are real hazards
54.
Treatment...
▪ The patienthaving previous hystory of
precipitate labour should be hospitalized
prior to labour
▪ During labour, the uterine contraction may
be suppressed by administering ether or
magnesium sulfate during contractions
▪ Delivery of the head should be
controlled
▪ Episiotomy should be done liberally
▪ Elective induction of labour by low rupture
of membranes and conduction of controlled
delivery is helpful
This type ofuterine contraction is
predominantly due to obstructed labour
Pathological anatomy of uterus...
▪There is gradual increase in intensity,
duration and frequency of uterine
contraction
▪The relaxation phase becomes less
and less, ultimately a state of tonic
contraction develops
▪ Retraction, however, continues
57.
▪ The lowersegment elongates and
becomes progressively thinner to
accomodate the fetus driven from the
upper segment
“ A circular groove encicling the uterus
is formed between the active upper
segment and the distended lower
segment, called pathological retraction
ring (Bandal's ring)”
▪ Due to pronounced retraction, there
is fetal jeopardy or even death
58.
▪ In primigravidae,further retraction
ceases in response to obstruction and
labor comes to a stand still a state of
uterine exhaustion
▪ Contractions may recommence after
a brief of rest with renewed vigour
▪ But in multipare, retraction
continues with progressive
circumferential dilatation and thinning
of the lower segment
59.
▪ There isprogressive rise of the
Bandal's ring, moving nearer and nearer
to the umbilicus and ultimately, the
lower segement ruptures
60.
Clinical features...
1. Patientis in agony from continuous pain
and discomfort and becomes restlessness
2. Features of exhaustion and ketoacidosis
are evident
3. Abdominal palpation reveals:
•Upper segment is harder and tender
•Lower segment is distended and tender
Treatment...
▪ Rupture ofthe uterus is to be excluded
▪ Internal version is contraindicated
▪ Correction of dehydration and ketoacidosis
by infusion of Ringer's solution
▪ Adequate pain relief
▪ Parenteral antibiotic ( Cefriaxone 1 g IV )
▪ Cesarean delivery is done in majority of
the cases
▪ Rupture of the uterus must be
excluded before attempting destructive
operation