1
BIOMECHANICS OF THORAX
AND CHEST WALL
2
 Most important
functions of the
thorax and chest
wall :
 Ventilation of
lungs
 Protection of
heart, lungs and
viscera
3
 Ventilatory mechanism in humans has three
parts
 The ribcage
 The ventilatory muscles
 The abdomen
4
5
Ribcage
 Truncated cone shaped
 Articulations of ribcage
 Manubriosternum
 Xiphisternum
 Costovertebral
 Costotransverse
 Costochondral
 Costosternal
 Interchondral
6
7
Manubriosternal and xiphisternal joints -
manibriumsternum
xiphisternum
Synchondrosis variety of
cartilaginous joints
8
 Sternal angle/angle of Louis corresponds to T4-T5
posteriorly.
 Bifurcation of trachea
 Aortic arch
9
Costovertebral joint
 Typical costoverterbral
joint - synovial type of
joint
 2nd to 9th ribs - typical
costoverterbral joint
 1st, 10th, 11th and 12th
atypical ribs articulate
only with their
corresponding vertebra
10
 The costovertebral joints are the articulations
that connect the heads of the ribs with the bodies
of the thoracic vertebrae. Joining of ribs to the
vertebrae occurs at two places, the head and the
tubercle of the rib. Two convex facets from the
head attach to two adjacent vertebrae. This forms a
trochoid joint, which is strengthened by the
ligament of the head and the intercapital
ligament.
11
Costotransverse joint
 Synovial joint
surrounded
by thin fibrous
capsule
 Is present from T1
to T10 vertebra and 1st
to 10th ribs
 This articulation is
reinforced by the
dorsal costotransverse
ligament
12
 Upper CT joints the
primary movement -
rotation
costal facet concave shape
and costal tubercle
convex shape
 T7 to T10 both articular
surfaces are flat and
gliding motion
predominates
13
 Costochondral joints are synchondroses with no
ligamentous support
 1st through 7th ribs articulate anterolaterally with
costal cartilages forming costochondral joint
14
 Costosternal (CS) joints
are formed by
attachment of the costal
cartilages of ribs to
sternum anteriorly
 CS joints of 1st, 6th and
7th ribs are
synchondroses
 CS joints of 2nd to 5th
ribs are synovial joints
15
Interchondral
joints
costosternal
 Interchondral joints are formed when 6th through
10th costal cartilages articulate with cartilage
above them and this is only way by which 8th to
10th ribs are connected to sternum
 Interchondral joints are synovial type of joints
16
 Costovertebral and costotransverse form a joint
couple mechanically linked
 Common movement - rotation about an axis
passing through centre of each joint for 1st to
10th ribs
 11th and 12th ribs it passes through the CV
joints as CT is absent for these two ribs
17
 Axis of motion - more towards frontal plane for
upper ribs and towards saggital plane for lower
ribs.
 During inspiration - ribs elevate, in upper ribs
motion simulates that of pump handle due to
frontal plane orientation of axis of motion, this
increases the anteroposterior diameter of
ribcage
18
 Axis of motion lying nearly in the saggital plane,
movement takes place more in lateral part in the
lower ribs
 Movement of 11th and 12th ribs varies from the
rest of the ribs
 Quadratus Lumborum muscle depresses and
fixes these ribs to provide adequate
diaphragmatic muscle tension
19
20
Axis towards frontal
plane
Axis towards saggital
plane
21
22
23
MUSCLES
 Muscles of the ribcage - ventilatory muscles
 Increased fatigue capacity and contract
rhythmically throughout life rather than
episodically
 Act primarily against the elastic properties of
lungs and airway resistance rather than
gravitational forces
24
 Neurological control of ventilatory muscles is
both voluntary and involuntary
 Recruitment of these muscles for ventilation
depends on the type of ventilation required
 Primary muscles of ventilation - Diaphragm,
Intercostals and scalenes
25
26
Diaphragm
27
 Diaphragm - accounts for 70% to 80% of quiet
breathing
 Functionally, muscle portion of diaphragm can be
divided as Costal and Crural parts
 Costal part - sternum, ribs (lower 6) and costal
cartilages
 Crural part - vertebral bodies (L1-L3)
28
29
Diaphragm contracts and pulls central tendon down
increasing vertical diameter of thorax
Decrease in the pleural pressure
Decrease in intrapulmonary pressure responsible for inspiration
30
 Costal fibers of diaphragm run vertically from
their origin close to the ribcage before getting
inserted into central tendon
 Portion of diaphragm which is close to inner wall
of lower ribcage - “ZONE OF APPOSIOTION”
 During tidal breathing, descent of dome of
diaphragm causes only slight change in its
shape, maintaining most of zone of apposition
31
32
 Crural portion has an indirect effect on
inspiration, it helps central tendon to descend
thus increasing pressure which is transmitted
through apposed diaphragm thus helping lower
ribcage to expand
33
34
 Diaphragm increases all three diameters of the
ribcage
Vertical diameter by contraction of central
tendon
Transverse diameter by elevating the lower ribs
Anteroposterior diameter by elevating the upper
ribs with the help of sternum
35
36
Intercostals
 Intercostal muscles - internal and external
intercostals
 Act as splints during inspiration and expiration, by
maintaining a constant tone
 Internal intercostal muscles run caudally and
posteriorly continuing dorsally where they become
posterior intercostal membrane at angle of the ribs
37
 External intercostals run caudally and at an
oblique angle to internal intercostals till
costochondral junction where they become
anterior intercostal membrane
38
 Anteriorly portion where only internal intercostals
are present - parasternal muscles
 Posteriorly only external intercostals are present
from the tubercle of ribs to the angle of ribs
 Laterally both external and internal intercostals
are present and are referred to as interosseous
or lateral intercostals
39
 Both set of intercostal muscles may be activated
during phases of respiration as minute ventilation
increases
 Activation of intercostals is from cephalic to
caudal end
40
41
 Parasternal muscles - primary inspiratory
muscles during quiet breathing and
stabilizers of ribcage
 Scalene are one of the primary muscles of quiet
respiration, their activity begins with onset of
inspiration and increases as inspiration gets
closer to total lung capacity
42
43
Accessory musclesof respiration
Inspiration
 Levatores costarum
 Pectoralis major/minor
 Rhomboids
 Anterior/medial/posterior
scalenes
 Serratus anterior and posterior
superior
 Subclavius, SCM
 Thoracic erector spinae
 Trapezius
Expiration
 Iliocostalis lumborum
 Transversus thoracis
Inspiration/Expiration
 Latissimus dorsi
 Quadratus lumborum
 Serratus posterior inferior
Maintenance of rib cage shape
 Intercostals
 Accessory muscles of ventilation
 When thorax is fixed, accessory muscles of
inspiration move vertebral column, arm or head on
the trunk
 Reverse muscle action
44
45
 Sternocleidomastoid flexes the cervical spine
when acting bilaterally
 When cervical spine is fixed the muscle moves
the ribcage superiorly, which expands upper
ribcage in pump handle mechanism
46
 Pectoralis major elevates upper ribcage when
shoulders and humerus are fixed. It can act both
as an inspiratory and an expiratory muscle
 Pectoralis minor can help raise third, fourth and
fifth ribs during a forced respiration
47
 Upper Trapezius can be helpful in active
inspiration in fixing the head so that
sternocledomastoid can act as a muscle of active
inspiration
 Subclavius is between the clavicle and first rib,
when acting in reverse action it can assist in
raising upper chest for inspiration
48
49
 Serratus anterior
 Serratus posterior
 Lat dorsi
 Sterno costalis
 Abdominal muscles –
 Tranversus abdominis
Internal abdominal oblique
External abdominal oblique
Rectus abdominis
50
Internal oblique
External oblique
Transverse
abdominis
51
52
Antagonismand synergismof diaphragm
and abdominal muscles :
 During inspiration – with diaphragmatic
contraction the central tendon descends down
increasing vertical thoracic diameter but this is
opposed by elongation of mediastinal elements
and also resistance of abdominal viscera.
At this time the abdominals relax allowing the abdomen
to bulge.
 This shows the synergistic activity of abdominals
 During expiration – diaphragm relaxes and
contraction of abdominal muscles lowers the
thoracic floor thereby decreasing simultaneously
the transverse and anteroposterior diameters of
thorax
 Also by pushing the viscera up abdominals
raise the central tendon
 This shows the antagonistic activity of
abdominals
53
54
 In normal breathing the respiratory muscles
should use less than 5% of the O2 taken in the
breath. If the diaphragm not working it is much
more.
55
56
Differencesassociated with neonates
 Healthy new born has an extremely compliant
chest wall because it is primarily cartilaginous
 Primary responsibility of ribcage stability on
ribcage muscles to counteract negative pleural
pressure of diaphragm during inspiration
 Ribs are also more horizontally placed this alters
angle of insertion of costal fibers of diaphragm
 Increased tendency of diaphragm fibers to pull
lower ribs inwards, thereby decreasing efficiency
of ventilation
 Only 20% of fibers of diaphragm are fatigue
resistance as compared to 50% in adults
 Accessory muscles are also at a disadvantage
57
58
Differencesassociated with elderly
 Pulmonary changes that occur may
affect pulmonary function
 Costal cartilages ossify, which interferes with
their axial rotation
 Many articulations undergo fibrosis with
advancing age
 Synovial joints undergo morphological changes
reducing mobility
59
 Chest wall compliance also reduces with age
 Lung tissue decreases in elasticity, thus
affecting elastic recoil property of lung and
outward pull of ribcage
 Results of these skeletal and tissue changes are
increase in functional residual capacity and
decrease in inspiratory capacity of thorax
 Loss of strength of skeletal muscles of
respiration
 Ventilatory muscles become more energy
expensive
 Resting position of diaphragm becomes less
domed with decrease in tone of abdominal
muscles
60
61
Scoliosis
 In scoliosis, if the curve is structural it
affects chest wall biomechanics and hence ventilation
 Lumbar and cervical curves cause minimal
change in chest wall biomechanics but a thoracic
curve causes restriction in ventilatory capacity,
restriction proportional to severity of curve
62
3 Types:
1. Nonstructural scoliosis
2. Transient structural scoliosis
3. Structural scoliosis (idiopathic accounts for 70–
80% of cases of scoliosis)
63
64
Kyphosis
An exaggeration of the normal posterior curve of the
spine.
 Results from change in structure and shape in
spine or posture.
 Fracture of anterior aspect of vertebral body –
Osteoporosis (OP).
 Scheuermann’s disease – Hereditary disorder that
results in kyphosis.
65
COPD
 In COPD, major manifestation is hyperinflation of
lungs due to destruction of alveolar walls
 Resting position of thorax in more of inspiratory
cycle against the normal resting position in
expiration
 This leads diaphragm to adapt a more flattened
configuration in its resting state rather than
acquiring its usual dome shape
 Flattened diaphragm will pull lower rib cage
inward, actually working against lung inflation
 This also decreases the zone of apposition
 Majority of inspiration performed by accessory
muscles, particularly the parasternal and scalene
66
Pectuscarinatum
67
Some patients develop a
rigid chest wall, in which
the AP diameter is
almost fixed in full
inspiration.
In these patients,
respiratory efforts are
less efficient.
68
 Vital capacity is reduced
 Residual air is increased
 Alveolar hypoventilation may ensue, with arterial
hypoxemia and the development of cor pulmonale.
 As the lungs lose compliance, incidence of
emphysema and frequency of infection are
increased.
Pectusexcavatum
69
Unless severe does not
cause restriction in
breathing , but due to
altered biomechanics
–shallow breathing
and dyspnea on
exertion may be
present
70
Flail chest
71
 The characteristic paradoxical motion of the flail
segment occurs due to pressure changes associated
with respiration that the rib cage normally resists:
 During normal inspiration, the diaphragm
contracts and intercostal muscles push the rib cage
out. Pressure in the thorax decreases below
atmospheric pressure, and air rushes in through
the trachea. However, a flail segment will not resist
the decreased pressure and will appear to push in
while the rest of the rib cage expands.
72
 During normal expiration, the diaphragm and
intercostal muscles relax, allowing the abdominal
organs to push air upwards and out of the thorax.
However, a flail segment will also be pushed out
while the rest of the rib cage contracts.
 The constant motion of the ribs in the flail
segment is painful, and, untreated, the sharp
broken edges of the ribs are likely to eventually
puncture the pleural sac and lung, possibly
causing a pneumothorax.
73
Paralysisof diaphragm/hemidiaphragm
74
Positioning
75
TREATMENT TECHNIQUES
76
Impaired Muscle Performance
Sources:
 Neurologic impairment or pathology
 Muscle strain or injury
 Disuse resulting in atrophy and general
deconditioning
 Length-associated changes resulting in altered
length-tension properties
77
Treatment
• Neural input must be restored for muscle performance
to improve.
• Protect weakened muscles from overstretch with
proper support.
• Stretch short muscles to maintain extensibility and
prevent contracture.
• For example: Impaired respiration – Stretch short
muscles and apply manual or elastic band resistance to
facilitate strength.
78
Stretch Lateral Trunk and
Intercostal Muscles
79
Muscle Strain or Injury
 Address posture and movement patterns.
 Improve performance of underused synergists.
 For example, in the case of overuse of anterior
scalene during breathing, reduce anterior scalene
use by improving performance of deep anterior
cervical flexors and instruct in proper pump and
bucket handle diaphragmatic breathing.
80
Disuse Resulting in Atrophyand General
Deconditioning
Caused by illness, immobilization, sedentary
lifestyle, subtle shifts in muscle balance.
Progressive resistive exercises for the upper body.
Initially, weight of limb is ample stimulus.
Progress in small increments.
Address balance between abdominal and spinal
extensors as well as thoracic multifidii.
81
Length-Associated Changes
Subtle imbalances in muscle length lead to length-
associated strength changes and positional
weakness of one synergist compared with agonist
or antagonist.
• Strengthen weak overstretched muscle groups in
shortened range.
• Stretch adaptively shortened muscles.
• Supportive taping adjunctive.
• Correction of posture and movement patterns.
82
Supportive Taping forThoracic Spine
83
Impaired ROM, Muscle Length, and Joint
Mobility/Integrity
Optimal function of the thoracic region requires
full symmetrical cardinal plane motion and full
rib motion.
Consider symmetrical breathing patterns.
Diagnose restrictions that are joint versus soft
tissue origin.
84
Establish the neutral spine
85
Exercise to PromoteThoracic Extension
86
Exercise Managementof Scoliosis
• Avoid symmetrical and spine flexibility exercises.
• Strengthen overstretched antagonist/synergist in
shortened range.
• Promote strength of the relatively weak muscle or
groups of muscles in the anterior thoracolumbar
region and the pelvic-hip complex.
• Trunk curl exercises or sit-ups are not indicated
methods of strengthening anterior thoracolumbar
muscles.
87
Thoracic Outlet Syndrome
• Characteristically young, slender women with
drooping shoulders and poor posture
• Treatment aimed at improving muscle
performance and reducing stretch to upper and
middle trapezius
• Supportive taping to elevate scapula
88
• Correct posture and movement relative to
neurovascular compression or stretching (i.e.,
depressed or anterior tilt scapula)
• Tape scapula into elevation to relieve compression
• Alter sleeping habits
• Improve diaphragmatic breathing
• Address associated physiologic/psychological
impairments
89
Summary
Functions
Joints
Movements
Muscles
Differences associated with neonates and elderly
Chest wall deformities
Treatment and correction of thoracic mechanics
90

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BIOMECHA OF THORAC AND RIB CAGE.pptx

  • 1. 1
  • 3.  Most important functions of the thorax and chest wall :  Ventilation of lungs  Protection of heart, lungs and viscera 3
  • 4.  Ventilatory mechanism in humans has three parts  The ribcage  The ventilatory muscles  The abdomen 4
  • 6.  Truncated cone shaped  Articulations of ribcage  Manubriosternum  Xiphisternum  Costovertebral  Costotransverse  Costochondral  Costosternal  Interchondral 6
  • 7. 7 Manubriosternal and xiphisternal joints - manibriumsternum xiphisternum Synchondrosis variety of cartilaginous joints
  • 8. 8  Sternal angle/angle of Louis corresponds to T4-T5 posteriorly.  Bifurcation of trachea  Aortic arch
  • 9. 9 Costovertebral joint  Typical costoverterbral joint - synovial type of joint  2nd to 9th ribs - typical costoverterbral joint  1st, 10th, 11th and 12th atypical ribs articulate only with their corresponding vertebra
  • 10. 10  The costovertebral joints are the articulations that connect the heads of the ribs with the bodies of the thoracic vertebrae. Joining of ribs to the vertebrae occurs at two places, the head and the tubercle of the rib. Two convex facets from the head attach to two adjacent vertebrae. This forms a trochoid joint, which is strengthened by the ligament of the head and the intercapital ligament.
  • 11. 11 Costotransverse joint  Synovial joint surrounded by thin fibrous capsule  Is present from T1 to T10 vertebra and 1st to 10th ribs  This articulation is reinforced by the dorsal costotransverse ligament
  • 12. 12  Upper CT joints the primary movement - rotation costal facet concave shape and costal tubercle convex shape  T7 to T10 both articular surfaces are flat and gliding motion predominates
  • 13. 13
  • 14.  Costochondral joints are synchondroses with no ligamentous support  1st through 7th ribs articulate anterolaterally with costal cartilages forming costochondral joint 14
  • 15.  Costosternal (CS) joints are formed by attachment of the costal cartilages of ribs to sternum anteriorly  CS joints of 1st, 6th and 7th ribs are synchondroses  CS joints of 2nd to 5th ribs are synovial joints 15 Interchondral joints costosternal
  • 16.  Interchondral joints are formed when 6th through 10th costal cartilages articulate with cartilage above them and this is only way by which 8th to 10th ribs are connected to sternum  Interchondral joints are synovial type of joints 16
  • 17.  Costovertebral and costotransverse form a joint couple mechanically linked  Common movement - rotation about an axis passing through centre of each joint for 1st to 10th ribs  11th and 12th ribs it passes through the CV joints as CT is absent for these two ribs 17
  • 18.  Axis of motion - more towards frontal plane for upper ribs and towards saggital plane for lower ribs.  During inspiration - ribs elevate, in upper ribs motion simulates that of pump handle due to frontal plane orientation of axis of motion, this increases the anteroposterior diameter of ribcage 18
  • 19.  Axis of motion lying nearly in the saggital plane, movement takes place more in lateral part in the lower ribs  Movement of 11th and 12th ribs varies from the rest of the ribs  Quadratus Lumborum muscle depresses and fixes these ribs to provide adequate diaphragmatic muscle tension 19
  • 20. 20 Axis towards frontal plane Axis towards saggital plane
  • 21. 21
  • 22. 22
  • 24.  Muscles of the ribcage - ventilatory muscles  Increased fatigue capacity and contract rhythmically throughout life rather than episodically  Act primarily against the elastic properties of lungs and airway resistance rather than gravitational forces 24
  • 25.  Neurological control of ventilatory muscles is both voluntary and involuntary  Recruitment of these muscles for ventilation depends on the type of ventilation required  Primary muscles of ventilation - Diaphragm, Intercostals and scalenes 25
  • 26. 26
  • 28.  Diaphragm - accounts for 70% to 80% of quiet breathing  Functionally, muscle portion of diaphragm can be divided as Costal and Crural parts  Costal part - sternum, ribs (lower 6) and costal cartilages  Crural part - vertebral bodies (L1-L3) 28
  • 29. 29
  • 30. Diaphragm contracts and pulls central tendon down increasing vertical diameter of thorax Decrease in the pleural pressure Decrease in intrapulmonary pressure responsible for inspiration 30
  • 31.  Costal fibers of diaphragm run vertically from their origin close to the ribcage before getting inserted into central tendon  Portion of diaphragm which is close to inner wall of lower ribcage - “ZONE OF APPOSIOTION”  During tidal breathing, descent of dome of diaphragm causes only slight change in its shape, maintaining most of zone of apposition 31
  • 32. 32
  • 33.  Crural portion has an indirect effect on inspiration, it helps central tendon to descend thus increasing pressure which is transmitted through apposed diaphragm thus helping lower ribcage to expand 33
  • 34. 34
  • 35.  Diaphragm increases all three diameters of the ribcage Vertical diameter by contraction of central tendon Transverse diameter by elevating the lower ribs Anteroposterior diameter by elevating the upper ribs with the help of sternum 35
  • 36. 36 Intercostals  Intercostal muscles - internal and external intercostals  Act as splints during inspiration and expiration, by maintaining a constant tone  Internal intercostal muscles run caudally and posteriorly continuing dorsally where they become posterior intercostal membrane at angle of the ribs
  • 37. 37  External intercostals run caudally and at an oblique angle to internal intercostals till costochondral junction where they become anterior intercostal membrane
  • 38. 38
  • 39.  Anteriorly portion where only internal intercostals are present - parasternal muscles  Posteriorly only external intercostals are present from the tubercle of ribs to the angle of ribs  Laterally both external and internal intercostals are present and are referred to as interosseous or lateral intercostals 39
  • 40.  Both set of intercostal muscles may be activated during phases of respiration as minute ventilation increases  Activation of intercostals is from cephalic to caudal end 40
  • 41. 41
  • 42.  Parasternal muscles - primary inspiratory muscles during quiet breathing and stabilizers of ribcage  Scalene are one of the primary muscles of quiet respiration, their activity begins with onset of inspiration and increases as inspiration gets closer to total lung capacity 42
  • 43. 43 Accessory musclesof respiration Inspiration  Levatores costarum  Pectoralis major/minor  Rhomboids  Anterior/medial/posterior scalenes  Serratus anterior and posterior superior  Subclavius, SCM  Thoracic erector spinae  Trapezius Expiration  Iliocostalis lumborum  Transversus thoracis Inspiration/Expiration  Latissimus dorsi  Quadratus lumborum  Serratus posterior inferior Maintenance of rib cage shape  Intercostals
  • 44.  Accessory muscles of ventilation  When thorax is fixed, accessory muscles of inspiration move vertebral column, arm or head on the trunk  Reverse muscle action 44
  • 45. 45
  • 46.  Sternocleidomastoid flexes the cervical spine when acting bilaterally  When cervical spine is fixed the muscle moves the ribcage superiorly, which expands upper ribcage in pump handle mechanism 46
  • 47.  Pectoralis major elevates upper ribcage when shoulders and humerus are fixed. It can act both as an inspiratory and an expiratory muscle  Pectoralis minor can help raise third, fourth and fifth ribs during a forced respiration 47
  • 48.  Upper Trapezius can be helpful in active inspiration in fixing the head so that sternocledomastoid can act as a muscle of active inspiration  Subclavius is between the clavicle and first rib, when acting in reverse action it can assist in raising upper chest for inspiration 48
  • 49. 49  Serratus anterior  Serratus posterior  Lat dorsi  Sterno costalis
  • 50.  Abdominal muscles –  Tranversus abdominis Internal abdominal oblique External abdominal oblique Rectus abdominis 50
  • 52. 52 Antagonismand synergismof diaphragm and abdominal muscles :  During inspiration – with diaphragmatic contraction the central tendon descends down increasing vertical thoracic diameter but this is opposed by elongation of mediastinal elements and also resistance of abdominal viscera. At this time the abdominals relax allowing the abdomen to bulge.  This shows the synergistic activity of abdominals
  • 53.  During expiration – diaphragm relaxes and contraction of abdominal muscles lowers the thoracic floor thereby decreasing simultaneously the transverse and anteroposterior diameters of thorax  Also by pushing the viscera up abdominals raise the central tendon  This shows the antagonistic activity of abdominals 53
  • 54. 54  In normal breathing the respiratory muscles should use less than 5% of the O2 taken in the breath. If the diaphragm not working it is much more.
  • 55. 55
  • 56. 56 Differencesassociated with neonates  Healthy new born has an extremely compliant chest wall because it is primarily cartilaginous  Primary responsibility of ribcage stability on ribcage muscles to counteract negative pleural pressure of diaphragm during inspiration  Ribs are also more horizontally placed this alters angle of insertion of costal fibers of diaphragm
  • 57.  Increased tendency of diaphragm fibers to pull lower ribs inwards, thereby decreasing efficiency of ventilation  Only 20% of fibers of diaphragm are fatigue resistance as compared to 50% in adults  Accessory muscles are also at a disadvantage 57
  • 58. 58 Differencesassociated with elderly  Pulmonary changes that occur may affect pulmonary function  Costal cartilages ossify, which interferes with their axial rotation  Many articulations undergo fibrosis with advancing age  Synovial joints undergo morphological changes reducing mobility
  • 59. 59  Chest wall compliance also reduces with age  Lung tissue decreases in elasticity, thus affecting elastic recoil property of lung and outward pull of ribcage  Results of these skeletal and tissue changes are increase in functional residual capacity and decrease in inspiratory capacity of thorax
  • 60.  Loss of strength of skeletal muscles of respiration  Ventilatory muscles become more energy expensive  Resting position of diaphragm becomes less domed with decrease in tone of abdominal muscles 60
  • 61. 61 Scoliosis  In scoliosis, if the curve is structural it affects chest wall biomechanics and hence ventilation  Lumbar and cervical curves cause minimal change in chest wall biomechanics but a thoracic curve causes restriction in ventilatory capacity, restriction proportional to severity of curve
  • 62. 62 3 Types: 1. Nonstructural scoliosis 2. Transient structural scoliosis 3. Structural scoliosis (idiopathic accounts for 70– 80% of cases of scoliosis)
  • 63. 63
  • 64. 64 Kyphosis An exaggeration of the normal posterior curve of the spine.  Results from change in structure and shape in spine or posture.  Fracture of anterior aspect of vertebral body – Osteoporosis (OP).  Scheuermann’s disease – Hereditary disorder that results in kyphosis.
  • 65. 65 COPD  In COPD, major manifestation is hyperinflation of lungs due to destruction of alveolar walls  Resting position of thorax in more of inspiratory cycle against the normal resting position in expiration  This leads diaphragm to adapt a more flattened configuration in its resting state rather than acquiring its usual dome shape
  • 66.  Flattened diaphragm will pull lower rib cage inward, actually working against lung inflation  This also decreases the zone of apposition  Majority of inspiration performed by accessory muscles, particularly the parasternal and scalene 66
  • 67. Pectuscarinatum 67 Some patients develop a rigid chest wall, in which the AP diameter is almost fixed in full inspiration. In these patients, respiratory efforts are less efficient.
  • 68. 68  Vital capacity is reduced  Residual air is increased  Alveolar hypoventilation may ensue, with arterial hypoxemia and the development of cor pulmonale.  As the lungs lose compliance, incidence of emphysema and frequency of infection are increased.
  • 69. Pectusexcavatum 69 Unless severe does not cause restriction in breathing , but due to altered biomechanics –shallow breathing and dyspnea on exertion may be present
  • 71. 71  The characteristic paradoxical motion of the flail segment occurs due to pressure changes associated with respiration that the rib cage normally resists:  During normal inspiration, the diaphragm contracts and intercostal muscles push the rib cage out. Pressure in the thorax decreases below atmospheric pressure, and air rushes in through the trachea. However, a flail segment will not resist the decreased pressure and will appear to push in while the rest of the rib cage expands.
  • 72. 72  During normal expiration, the diaphragm and intercostal muscles relax, allowing the abdominal organs to push air upwards and out of the thorax. However, a flail segment will also be pushed out while the rest of the rib cage contracts.  The constant motion of the ribs in the flail segment is painful, and, untreated, the sharp broken edges of the ribs are likely to eventually puncture the pleural sac and lung, possibly causing a pneumothorax.
  • 76. 76 Impaired Muscle Performance Sources:  Neurologic impairment or pathology  Muscle strain or injury  Disuse resulting in atrophy and general deconditioning  Length-associated changes resulting in altered length-tension properties
  • 77. 77 Treatment • Neural input must be restored for muscle performance to improve. • Protect weakened muscles from overstretch with proper support. • Stretch short muscles to maintain extensibility and prevent contracture. • For example: Impaired respiration – Stretch short muscles and apply manual or elastic band resistance to facilitate strength.
  • 78. 78 Stretch Lateral Trunk and Intercostal Muscles
  • 79. 79 Muscle Strain or Injury  Address posture and movement patterns.  Improve performance of underused synergists.  For example, in the case of overuse of anterior scalene during breathing, reduce anterior scalene use by improving performance of deep anterior cervical flexors and instruct in proper pump and bucket handle diaphragmatic breathing.
  • 80. 80 Disuse Resulting in Atrophyand General Deconditioning Caused by illness, immobilization, sedentary lifestyle, subtle shifts in muscle balance. Progressive resistive exercises for the upper body. Initially, weight of limb is ample stimulus. Progress in small increments. Address balance between abdominal and spinal extensors as well as thoracic multifidii.
  • 81. 81 Length-Associated Changes Subtle imbalances in muscle length lead to length- associated strength changes and positional weakness of one synergist compared with agonist or antagonist. • Strengthen weak overstretched muscle groups in shortened range. • Stretch adaptively shortened muscles. • Supportive taping adjunctive. • Correction of posture and movement patterns.
  • 83. 83 Impaired ROM, Muscle Length, and Joint Mobility/Integrity Optimal function of the thoracic region requires full symmetrical cardinal plane motion and full rib motion. Consider symmetrical breathing patterns. Diagnose restrictions that are joint versus soft tissue origin.
  • 86. 86 Exercise Managementof Scoliosis • Avoid symmetrical and spine flexibility exercises. • Strengthen overstretched antagonist/synergist in shortened range. • Promote strength of the relatively weak muscle or groups of muscles in the anterior thoracolumbar region and the pelvic-hip complex. • Trunk curl exercises or sit-ups are not indicated methods of strengthening anterior thoracolumbar muscles.
  • 87. 87 Thoracic Outlet Syndrome • Characteristically young, slender women with drooping shoulders and poor posture • Treatment aimed at improving muscle performance and reducing stretch to upper and middle trapezius • Supportive taping to elevate scapula
  • 88. 88 • Correct posture and movement relative to neurovascular compression or stretching (i.e., depressed or anterior tilt scapula) • Tape scapula into elevation to relieve compression • Alter sleeping habits • Improve diaphragmatic breathing • Address associated physiologic/psychological impairments
  • 89. 89 Summary Functions Joints Movements Muscles Differences associated with neonates and elderly Chest wall deformities Treatment and correction of thoracic mechanics
  • 90. 90

Editor's Notes

  • #10: The costovertebral joints are the articulations that connect the heads of the ribs with the bodies of the thoracic vertebrae. Joining of ribs to the vertebrae occurs at two places, the head and the tubercle of the rib. Two convex facets from the head attach to two adjacent vertebrae. This forms a trochoid joint, which is strengthened by the ligament of the head and the intercapital ligament. Articulation of the tubercle is to the transverse process of the adjacent vertebrae. This articulation is reinforced by the dorsal costotransverse ligament.
  • #70: Add flail chest and symmetric and asymmetric chest wall abnormalities